e-learning
resources
Amsterdam 2015
Monday, 28.09.2015
Respiratory infections: from basic science to clinical issues
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
LSC Abstract – Chitinase-like protein Chil1 modulates type I interferon response through TLR9-AP3-IRF7 axis during streptococcus pneumoniae lung infection
Charles Dela Cruz (New Haven, United States of America), Wei Liu, Chad Marion, Rajvir Singh, Chuan Hua He, Chun Geun Lee, Jack Elias, Charles Dela Cruz
Source:
International Congress 2015 – Respiratory infections: from basic science to clinical issues
Session:
Respiratory infections: from basic science to clinical issues
Session type:
Thematic Poster Session
Number:
2670
Disease area:
Respiratory infections
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Charles Dela Cruz (New Haven, United States of America), Wei Liu, Chad Marion, Rajvir Singh, Chuan Hua He, Chun Geun Lee, Jack Elias, Charles Dela Cruz. LSC Abstract – Chitinase-like protein Chil1 modulates type I interferon response through TLR9-AP3-IRF7 axis during streptococcus pneumoniae lung infection. Eur Respir J 2015; 46: Suppl. 59, 2670
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Management of severe community acquired pneumonia – ERS guidelines
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Predictive indexes in prolonged weaning of mechanical ventilation due to tracheostomy in patients with COVID-19 pneumonia
Related content which might interest you:
Chlamydia pneumoniae enhances infection with Haemophilus influenzae, up-regulates TLR2-expression in human lung tissue and leads to enhanced chemokine production after coinfection – a possible mechanism for COPD exacerbations
Source: Annual Congress 2008 - New diagnostic methods in viral and atypical infections
Year: 2008
Interaction between
streptococcus pneumoniae
and human lung cells in an acute tissue infection model
Source: Eur Respir J 2006; 28: Suppl. 50, 577s
Year: 2006
Chlamydia pneumoniae
persists in COPD lung tissue: predominant role of toll-like receptor 2 versus 4 in
chlamydia pneumoniae
-induced activation of pulmonary immune cells
Source: Eur Respir J 2004; 24: Suppl. 48, 240s
Year: 2004
LSC 2014 abstract - Proteinase activated receptor-1 (PAR1) plays a key role in the early inflammatory response to streptococcus pneumoniae pulmonary infection
Source: International Congress 2014 – Immunology of respiratory infections
Year: 2014
LSC Abstract – The inflammatory response to streptococcus pneumoniae is exaggerated by the polysaccharide capsule
Source: International Congress 2015 – Respiratory infections: from basic science to clinical issues
Year: 2015
Increased Th2 response with
C. pneumoniae
infection
Source: Eur Respir J 2002; 20: Suppl. 38, 311s
Year: 2002
Club cell secretory protein: a key mediator in Mycoplasma pneumoniae infection.
Source: International Congress 2018 – Disease exacerbations from a molecular point of view
Year: 2018
microRNAs constitute a negative feedback loop in streptococcus pneumoniae induced macrophage activation
Source: International Congress 2016 – Airway response to infection
Year: 2016
Nontypeable Haemophilus influenzae infection of human lung tissue induces proinflammatory cytokine response via toll-like receptor 2 and EGF-receptor
Source: Annual Congress 2008 - Mechanisms of respiratory infections: interaction between the pathogen and the host
Year: 2008
Differential regulation of Moraxella catarrhalis-induced interleukin-8 response by protein kinase C isoforms
Source: Eur Respir J 2008; 31: 725-735
Year: 2008
TLR9- and Src-dependent expression of Krueppel-like factor 4 controls interleukin-10 expression in pneumonia
Source: Eur Respir J 2013; 41: 384-391
Year: 2013
Osteopontin promotes host defense during Klebsiella pneumoniae-induced pneumonia
Source: Eur Respir J 2010; 36: 1337-1345
Year: 2010
Overexpression of macrophage inducible C-type lectin Mincle aggravates proinflammatory responses to Streptococcus pneumoniae with fatal outcome in mice
Source: Virtual Congress 2020 – Translational science in respiratory infections
Year: 2020
Antiapoptotic and proinflammatory function of toll-like receptor 2 in acute
C. pneumoniae
infection of COPD lung tissue
Source: Eur Respir J 2006; 28: Suppl. 50, 575s
Year: 2006
Immunokinetics in severe pneumonia due to influenza virus and bacteria coinfection in mice
Source: Eur Respir J 2004; 24: 143-149
Year: 2004
The impact of time on the systemic inflammatory response in pneumococcal pneumonia
Source: Eur Respir J 2010; 35: 614-618
Year: 2010
Streptococcus pneumoniae augments inflammatory process in asthma model without changing Th2 profile
Source: Virtual Congress 2020 – Monitoring of inflammation in remodelling
Year: 2020
Host immune response to Chlamydia pneumoniae heat shock protein 60 is associated with asthma
Source: Eur Respir J 2001; 17: 1078-1082
Year: 2001
Streptococcus pneumoniae infection suppresses allergic airways disease by inducing regulatory T-cells
Source: Eur Respir J 2011; 37: 53-64
Year: 2011
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept