e-learning
resources
Munich 2014
Sunday, 07.09.2014
Animal models: asthma, COPD and beyond
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Allergic asthma induce opposite changes in the expression of caveolin-1 and cavin-2 in airway and vascular pulmonary smooth muscle
S. L. Sánchez Hernández, M. álvarez Santos, F. Gutiérrez Aguilar, F. ávila Carbajal, N. Bobadilla, R. Lascurain, G. Gutiérrez Olguín, B. Bazán Perkins (Mexico City, Mexico)
Source:
International Congress 2014 – Animal models: asthma, COPD and beyond
Session:
Animal models: asthma, COPD and beyond
Session type:
Thematic Poster Session
Number:
858
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
S. L. Sánchez Hernández, M. álvarez Santos, F. Gutiérrez Aguilar, F. ávila Carbajal, N. Bobadilla, R. Lascurain, G. Gutiérrez Olguín, B. Bazán Perkins (Mexico City, Mexico). Allergic asthma induce opposite changes in the expression of caveolin-1 and cavin-2 in airway and vascular pulmonary smooth muscle. Eur Respir J 2014; 44: Suppl. 58, 858
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Corticosteroid insensitivity in airway smooth muscle cells from severe asthma is dependent on stimulus and cytokine product
Source: International Congress 2014 – Cell biology 2014
Year: 2014
CCL2 release by airway smooth muscle is increased in asthma and promotes fibrocyte migration
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Airway smooth muscle myosin phosphatase target subunit 1 phosphorylation in allergic asthma model
Source: International Congress 2014 – Animal models: asthma, COPD and beyond
Year: 2014
The role of glycolysis in TGF-beta-mediated airway smooth muscle cell proliferation
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Hypoxia exerts dualistic effects on inflammatory and proliferative responses of healthy and asthmatic primary human bronchial smooth muscle cells
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Mitochondrial membrane potential in the airway and skeletal muscle compartments of smokers with and without COPD
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
Reconstituted human airway epithelium 3D-model to assess the impact of air pollutants on the inflammatory response
Source: International Congress 2014 – Mechanistic studies of the airway epithelium
Year: 2014
Link between airway inflammation and remodeling: TNFα induces airway smooth muscle cell proliferation via an ET1/GMCSF/IL6 network
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015
Omalizumab prevents smooth muscle cell growth induced by asthmatic serum
Source: International Congress 2014 – New mechanisms in the pathogenesis of asthma and other lung diseases
Year: 2014
Enhanced inflammatory gene expression in bronchial epithelial cells from asthmatic patients: Attenuation by a Brd4 mimic
Source: International Congress 2014 – Novel targets and drugs for asthma and COPD
Year: 2014
Secretion of hyaluronic acid is mediated by muscarinic receptors in airway smooth muscle cells of patients with COPD
Source: International Congress 2014 – Translational studies in COPD
Year: 2014
The role of the K
Ca
3.1 channel in the regulation of airway mucus hypersecretion in asthma
Source: International Congress 2014 – Mechanistic studies of the airway epithelium
Year: 2014
Targeting ASK1 in preventing airway smooth muscle growth: Implications for airway remodeling in COPD
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
Integrins regulate eosinophil adhesion to the airway smooth muscle cells in asthma patients
Source: International Congress 2015 – Asthma mechanisms and management
Year: 2015
Potential role of SPARC, a downstream mediator of TGF-b in chronic airways disease
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015
Investigating mitochondrial dysfunction in asthma
Source: International Congress 2015 – Advances in the future treatment of severe asthma
Year: 2015
S100 proteins induce MUC5AC production in airway epithelial cells
Source: International Congress 2014 – Mechanistic studies of the airway epithelium
Year: 2014
Effects of aerobic training on perivascular inflammation and on vascular activation in a model of asthma
Source: International Congress 2014 – Animal models: asthma, COPD and beyond
Year: 2014
Association between airway inflammation, airway remodelling and physiological changes in asthma
Source: International Congress 2014 – New mechanisms in the pathogenesis of asthma and other lung diseases
Year: 2014
Eosinophil-induced airway smooth muscle cells remodeling is attenuated by blocking of RGD-binding integrins in asthma
Source: International Congress 2016 – New findings in mucosal immunology
Year: 2016
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept