Immunohistochemical localization of vanilloid receptor subtype 1 (TRPV1) in the guinea pig respiratory system Source: Eur Respir J 2004; 24: Suppl. 48, 554s Year: 2004
Modulatory role of tachykinin NK1 receptor in cholinergic contraction of mouse trachea Source: Eur Respir J 2003; 21: 3-10 Year: 2003
Ethanol (EtOH) enhances the transient receptor potential vanilloid 1 (TRPV1) dependent cough in the guinea pig Source: Eur Respir J 2004; 24: Suppl. 48, 94s Year: 2004
Ethanol (EtOH) promotes Ca2+ mobilization and neuropeptide release from airway sensory nerves via vanilloid receptor 1 (VR1) activation Source: Eur Respir J 2002; 20: Suppl. 38, 170s Year: 2002
Modulation of transient receptor potential vanilloid-1 (TRPV1) by inhaled prostaglandin-E2 (PGE2) and bradykinin (BK) is associated with increased cough sensitivity to capsaicin (CPS) and autonomic dysregulation of cardiac rhythm in healthy subjects Source: Virtual Congress 2020 – Occupational and environmental causes of respiratory disease Year: 2020
Hydrogen sulphide (H2 S) modulates cough in the guinea pig activating/sensitising the transient receptor potential vanilloid 1 (TRPV1) Source: Eur Respir J 2005; 26: Suppl. 49, 130s Year: 2005
The effect of leukotrienes and their receptor antagonists on calcium signalling in the cough receptors transient receptor potential ankyrin 1 (TRPA1) and vanilloid 1 (TRPV1) Source: Annual Congress 2010 - Models of airways disease Year: 2010
Ethanol (EtOH) causes bronchoconstriction and plasma protein extravasation (PPE) in rodent airways via vanilloid receptor 1 (VR1) activation Source: Eur Respir J 2002; 20: Suppl. 38, 485s Year: 2002
CS-003, a novel triple neurokinin receptor antagonist, inhibits symptoms of respiratory diseases models in guinea pigs Source: Eur Respir J 2006; 28: Suppl. 50, 664s Year: 2006
Different alcohols activate airway sensory nerves by a vanilloid receptor 1 (TRPV1) dependent mechanism Source: Eur Respir J 2003; 22: Suppl. 45, 415s Year: 2003
Protease-activated receptor 2 (PAR2) stimulation potentiates citric acid induced cough by sensitising the vanilloid receptor-1 (TRPV1) Source: Annual Congress 2003 - Novel drugs under development for the treatment of respiratory diseases Year: 2003
Combined loss of M2 and M3 muscarinic acetylcholine receptor (mAChR) function in mutant mice increases serotonin airway responsiveness (AR) Source: International Congress 2014 – Animal models in respiratory drug development Year: 2014
Nerve growth factor induces the expression of vanilloid receptor 1 in airway sensory neurons Source: Eur Respir J 2002; 20: Suppl. 38, 385s Year: 2002
Nicotinic alpha-7 receptor stimulation (a7nAChR) inhibited NF-kB/STAT3/SOCS3 pathways in a murine model of asthma. Source: International Congress 2017 – Novel mechanisms and treatments for asthma Year: 2017
A new ultra-potent vanilloid receptor 1 (VR1) antagonist abates capsaicin and citric acid induced cough Source: Eur Respir J 2002; 20: Suppl. 38, 190s Year: 2002
The cannabinoid receptor agonist, WIN 55,212-2, inhibits antigen-induced plasma extravasation in guinea pig airways Source: Annual Congress 2010 - Models of airways disease Year: 2010
Activation of transient receptor potential (TRP) channels by hypoosmolar solution: an endogenous mechanism of ATP release and afferent nerve activation Source: International Congress 2017 – Novel studies on bronchodilators and cough Year: 2017
The endocannabinoids anandamide and 2-arachidonyl-glycerol (2-AG) induce pulmonary hypertension (PH) in a CB1 receptor independent manner - role of COX-2 and EP1 receptor activation Source: Eur Respir J 2005; 26: Suppl. 49, 733s Year: 2005
EP2 & EP4 receptor agonists inhibit the airway sensory nerves that cause cough, a potential protective control that is lost in human smokers and a pre-clinical COPD model Source: International Congress 2017 – Novel mechanisms and treatments for COPD Year: 2017
Serotonin (5-hydroxytryptamine) modulates cytokine and chemokine production in lipopolysaccharide-primed human monocytes via stimulation of different 5-HT receptor subtypes Source: Annual Congress 2005 - Inflammation and remodelling in allergy and asthma Year: 2005