Source: Annual Congress 2013 –Genetic factors in airway development and disease
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
M. Perry, P. Austin, I. Adcock, K. Fan Chung (London, United Kingdom). LATE-BREAKING ABSTRACT: The long intergenic non-coding RNA, PVT1, regulates the TGF-beta-induced proliferative response in airway smooth muscle cells from severe asthmatics. Eur Respir J 2013; 42: Suppl. 57, 1520

Member's Comments

No comment yet.

Related content which might interest you: Impaired inhibitory action of corticosteroids on chemokine expression induced by TNFalpha in airway smooth muscle (ASM) cells from patients with severe asthma Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Long intergenic non-coding RNAs are involved in the differentiation of smooth muscle cells Source: Annual Congress 2013 –Pulmonary circulation: basic science Year: 2013 DP2/CRTh2 is expressed by ASM cells in asthma and its inhibition suppresses ASM migration Source: International Congress 2016 – Mechanisms of disease Year: 2016 WNT-5A and WNT-11 as novel regulators of the contractile airway smooth muscle phenotype Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Compound A (CpdA) suppressed production of corticosteroid-resistant chemokines via GR-independent mechanisms in airway smooth muscle (ASM) cells Source: Annual Congress 2013 –Preclinical models for the development of new drugs for respiratory diseases Year: 2013 MNK-1 inhibition reduces proliferation and CXCL10 in airway smooth muscle cells Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 FGF-2 modulates human airway smooth muscle contractile protein expression and cell stiffness Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Reduced MicroRNA-19a causes constitutive high expression of PRMT1 and remodeling in asthma Source: International Congress 2016 – New therapies and therapeutic targets Year: 2016 p53 dysfunction increased mitochondrial biogenesis and bronchial smooth muscle cell proliferation in asthma Source: International Congress 2015 – Airway remodelling: recent developments Year: 2015 Plasminogen-stimulated inflammatory cytokine production by airway smooth muscle cells is regulated by annexin A2 Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Effect of oxidative stress on mitochondrial function in airway smooth muscle (ASM) cells from COPD patients Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Role of protein phosphatase 5 (PP5) in mediating corticosteroid insensitivity in airway smooth muscle (ASM) cells in severe asthma Source: International Congress 2015 – New insights into lung disease pathogenesis Year: 2015 HMGB1 is upregulated in ASM in asthma and induces ASM contraction via TLR4 Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects Year: 2015 Compound A modulates steroid insensitive chemokines in airway smooth muscle cells via IRF-1 dependent and independent pathways Source: International Congress 2015 – New targets and modalities for the treatment of asthma and COPD Year: 2015 Cigarette smoke extract and TGF-beta1 induce distinctive expression of extracellular matrix protein genes in human airway smooth muscle cells Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation Year: 2013 A functional role for WNT-5A in driving airway myocyte proliferation Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation Year: 2013 TGF-beta alters TRPC6 expression and calcium handling via smad2/3 in human airway smooth muscle Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Eosinophil-induced airway smooth muscle cells remodeling is attenuated by blocking of RGD-binding integrins in asthma Source: International Congress 2016 – New findings in mucosal immunology Year: 2016 The extracellular matrix deposited by asthmatic airway smooth muscle cells in a resting state reflects a healthy matrix Source: International Congress 2015 – Airway remodelling: recent developments Year: 2015 Upregulation of airway smooth muscle (ASM) cell apoptosis may limit ASM growth during airway remodeling in experimental asthma Source: Annual Congress 2008 - Novel mechanisms in the pathogenesis of pulmonary inflammation Year: 2008