e-learning
resources
Barcelona 2013
Sunday, 08.09.2013
Novel mechanisms in lung injury
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
High-stretch, but not atelectasis, causes systemic cytokine release by lung-marginated monocytes
K. Wakabayashi, M. Wilson, K. Tatham, K. O'Dea, M. Takata (London, United Kingdom)
Source:
Annual Congress 2013 –Novel mechanisms in lung injury
Session:
Novel mechanisms in lung injury
Session type:
Thematic Poster Session
Number:
653
Disease area:
Respiratory critical care
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
K. Wakabayashi, M. Wilson, K. Tatham, K. O'Dea, M. Takata (London, United Kingdom). High-stretch, but not atelectasis, causes systemic cytokine release by lung-marginated monocytes. Eur Respir J 2013; 42: Suppl. 57, 653
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
From pulmonary gas exchange to the interpretation of arterial blood gases
The physiology of breathlessness
Respiratory physiology
Related content which might interest you:
New IL-1 family IL-38 is highly expressed in alveolar cells of drug-induced lung injury and idiopathic pulmonary fibrosis
Source: International Congress 2016 – ILD pathogenesis
Year: 2016
A mouse model of acute lung injury implicates phospholipase Cε in neutrophilic inflammation through CXC chemokine production
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
Microvesicles derived from alveolar macrophages mediate epithelial activation via a TNF dependant mechanism
Source: International Congress 2015 – Novel insights into the pathobiology of acute lung injury
Year: 2015
Airway inflammation during stable and acutely exacerbated chronic obstructive pulmonary disease
Source: Eur Respir J 2005; 25: 640-646
Year: 2005
ATP selectively packages TNF within microvesicles released from alveolar macrophages
Source: International Congress 2016 – Novel insights into alveolar and bronchial epithelial cell injury and repair
Year: 2016
Pirfenidone suppresses proinflammatory and antiangiogenetic cytokine production by alveolar macrophages (AMs) in idiopathic non specific interstitial pneumonia (iNSIP)
Source: International Congress 2014 – ILDs 2
Year: 2014
The role of ATP and purinergic-receptor- signalling in the pathogenesis of ALI/ARDS
Source: Annual Congress 2013 –Cell signalling and inflammation: what's new in 2013?
Year: 2013
Dexamethasone inhibits secretory and cytosolic phospholipases A2 in alveolar macrophages in acute lung injury
Source: Annual Congress 2013 –Macrophages, mononuclear cells and viral infection
Year: 2013
Heparin effect in alveolar cells and macrophages in an acute lung injury model
Source: International Congress 2015 – Novel insights into the pathobiology of acute lung injury
Year: 2015
Cellular origin of pro-coagulant and (anti)-fibrinolytic factors in bleomycin-injured lungs
Source: Eur Respir J 2007; 29: 1105-1114
Year: 2007
TNF and IP-10 drive inflammation into hyperinflammation in the murine lung
Source: Annual Congress 2013 –The role of hormones and cytokines in lung injury
Year: 2013
Effect of nintedanib on the release of angiogenic/angiostatic cytokines by alveolar macrophages (AMs) in interstitial lung diseases (ILD)
Source: International Congress 2015 – IPF: from bench to bedside
Year: 2015
Airway neutrophilia in COPD is not associated with increased neutrophil survival
Source: Eur Respir J 2006; 28: 1163-1169
Year: 2006
Down-regulation of RIP140 can prevent LPS-induced inflammation and acute lung injury in mice via PPARγ
Source: International Congress 2015 – Novel insights into the pathobiology of acute lung injury
Year: 2015
Effects of ceramide pathway inhibition on the inflammatory response in lipopolysacharide-triggered lung injury
Source: International Congress 2015 – Respiratory muscles and airways function: from physiology to clinical implications
Year: 2015
Resolution of acute lung injury and inflammation: a translational mouse model
Source: Eur Respir J 2012; 39: 1162-1170
Year: 2012
Inhibition of acute lung inflammation by removal of pulmonary intravascular macrophages (PIMs)
Source: Eur Respir J 2001; 18: Suppl. 33, 79s
Year: 2001
Neutrophilia is associated with the persistent parenchymal changes after acute lung injury
Source: International Congress 2015 – Latest insights into chronic care
Year: 2015
Impaired Alveolar Macrophage Efferocytosis in ARDS causes accumulation of apoptotic neutrophils & prolonged inflammation
Source: International Congress 2018 – Role of microbial exposure and inflammation in lung injury
Year: 2018
Thalidomide reduces IL-18, IL-8 and TNF-α release from alveolar macrophages in interstitial lung disease
Source: Eur Respir J 2006 Oct 01;28(4):824-831
Year: 2006
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept