e-learning
resources
Vienna 2012
Sunday, 02.09.2012
The many roads to lung injury
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Remodelling of cytoskeleton towards a softer cell by latrunculin prevents stretch-induced apoptosis in alveolar type II cells
H. Kuhn, T. Fuchs, F. Wetzel, S. Hammerschmidt, J.A. Käs, H. Wirtz (Leipzig, Germany)
Source:
Annual Congress 2012 - The many roads to lung injury
Session:
The many roads to lung injury
Session type:
Thematic Poster Session
Number:
820
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
H. Kuhn, T. Fuchs, F. Wetzel, S. Hammerschmidt, J.A. Käs, H. Wirtz (Leipzig, Germany). Remodelling of cytoskeleton towards a softer cell by latrunculin prevents stretch-induced apoptosis in alveolar type II cells. Eur Respir J 2012; 40: Suppl. 56, 820
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Impact of Dexamethasone on pathogen profile of COVID-19 patients requiring intensive care: a multicentre retrospective study
Muscle energy techniques for COPD patients: Effects on pulmonary function and activities of daily living
Related content which might interest you:
Mechanical stretch induced apoptosis and non-apoptotic cell death in alveolar type II cells and its modulation by captopril and L-arginin
Source: Eur Respir J 2003; 22: Suppl. 45, 587s
Year: 2003
Apoptosis and non-apoptotic cell death induced by mechanical stress in alveolar type II cells and pulmonary microvascular endothelial cells
Source: Eur Respir J 2003; 22: Suppl. 45, 587s
Year: 2003
Influence of protein kinase C on stretch-induced apoptosis in rat alveolar type II cells
Source: Annual Congress 2012 - The many roads to lung injury
Year: 2012
Inhibition of bradykinin breakdown by ACE inhibitors reduces mechanical stretch-induced apoptosis of alveolar type II cells
Source: Eur Respir J 2005; 26: Suppl. 49, 345s
Year: 2005
Cell stiffening and contraction induced by thrombin in alveolar epithelial cells treated with dexamethasone
Source: Eur Respir J 2006; 28: Suppl. 50, 831s
Year: 2006
Autocrine secretion of bradykinin prevents alveolar type II cells from mechanical stretch-induced apoptosis by activation – role of PI3K and Akt
Source: Annual Congress 2008 - Border patrol: the alveolar barrier in lung injury
Year: 2008
Effects of static stretch on secretory function and morphology in alveolar epithelial type II cells
Source: Annual Congress 2008 - Border patrol: the alveolar barrier in lung injury
Year: 2008
High concentrations of VEGF reduce stretch-induced apoptosis of alveolar type II cells
Source: Annual Congress 2009 - Highlighting the epithelial cell in lung disease: from COPD to cancer to acute lung injury
Year: 2009
Actin cytoskeleton and myosin light chain (MLC) phosphorylation modulate thrombin-induced contraction of alveolar epithelial cells
Source: Eur Respir J 2005; 26: Suppl. 49, 714s
Year: 2005
Surface expression of CD74 by type II alveolar epithelial cells: a potential mechanism for macrophage migration inhibitory factor-induced epithelial repair
Source: Annual Congress 2008 - Border patrol: the alveolar barrier in lung injury
Year: 2008
Hyperoxia reversibly inhibits the transdifferentiation of alveolar type II (ATII) cells to alveolar type I (ATI) cells
Source: Annual Congress 2005 - Cellular and molecular approaches to lung injury research
Year: 2005
Effect of activated protein C on cell contraction of human alveolar epithelial cells subjected to thrombin
Source: Annual Congress 2008 - Pathophysiology of acute lung injury
Year: 2008
Wnt11 inhibits epithelial-mesenchymal transition induced by TGFb1 in human type II alveolar epithelial cells
Source: Annual Congress 2012 - Fibrogenesis between epithelial injury and fibroblast proliferation
Year: 2012
RANKL contributes to lung tissue repair via promoting type II epithelial cell proliferation
Source: Virtual Congress 2020 – Elucidating the mechanisms underlying lung repair and exploring therapeutic strategies in vitro
Year: 2020
PP126 – RANKL contributes to lung tissue repair via promoting type II epithelial cell proliferation
Source: ERS Lung Science Conference 2021
Year: 2021
The growth factors HGF and KGF prevent mechanical stretch-induced apoptosis of alveolar Type II cells by restoration of mechanical stretch-induced decreased phosphatidylinositol 3-OH kinase (PI3K) activity
Source: Eur Respir J 2006; 28: Suppl. 50, 568s
Year: 2006
PTEN deficiency in alveolar type 2 cells accelerates cellular senescence, disrupts epithelial–mesenchymal and mitochondrial homeostasis, and induces age-related emphysema
Source: International Congress 2019 – Cellular metabolism and immune senescence in lung pathology
Year: 2019
TGFβ1-induced extracellular matrix production enhances airway smooth muscle cell proliferation
Source: Annual Congress 2011 - Airway smooth muscle cells and fibroblasts: cell biology
Year: 2011
Effect of hypoxia on gene regulation and differentiation in alveolar epithelial type II cells
Source: Eur Respir J 2001; 18: Suppl. 33, 162s
Year: 2001
Hydrogen sulfide inhibits cell proliferation and induces cell cycle arrest in airway smooth muscle cells
Source: Annual Congress 2010 - Models of airways disease
Year: 2010
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept