Tobacco smoking alters the relationship between airway inflammation and airway hyperresponsiveness in asthma

C.G. Westergaard, C. Porsbjerg, V. Backer (Copenhagen, Denmark)

Source: Annual Congress 2012 - Cell biology and inflammatory gene expression in chronic lung disease
Session: Cell biology and inflammatory gene expression in chronic lung disease
Session type: Thematic Poster Session
Number: 808
Disease area: Airway diseases

Congress or journal article abstract

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
C.G. Westergaard, C. Porsbjerg, V. Backer (Copenhagen, Denmark). Tobacco smoking alters the relationship between airway inflammation and airway hyperresponsiveness in asthma. Eur Respir J 2012; 40: Suppl. 56, 808

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Smoking effect on airways inflammation in asthma and COPD: relationship with airways reversibility and hyperresponsiveness
Source: Annual Congress 2009 - COPD: from biomarker profiling to clinical assessment
Year: 2009


Effects of active and passive smoking on allergic airway inflammation and airway hyperresponsiveness in mice
Source: Eur Respir J 2002; 20: Suppl. 38, 310s
Year: 2002

Airway remedeling in a mouse model of chronic asthma: relation between the degree of airway remodeling and airway hyperresponsiveness
Source: Eur Respir J 2004; 24: Suppl. 48, 38s
Year: 2004

Smoking cessation attenuates AMP but not methacholine airway hyperresponsiveness in heavy smokers
Source: Annual Congress 2006 - New investigational approaches to smoking and smoking cessation
Year: 2006


Time domain and flow indices of bronchial hyperresponsiveness: association with asthma symptoms, atopy and smoking
Source: Eur Respir J 2002; 20: 86-91
Year: 2002



Smoking cessation improves both direct and indirect airway hyperresponsiveness in COPD
Source: Eur Respir J 2004; 24: 391-396
Year: 2004



Increased airway closure is a determinant of airway hyperresponsiveness
Source: Eur Respir J 2008; 32: 1563-1569
Year: 2008



Dose-dependent association of smoking and bronchial hyperresponsiveness
Source: Eur Respir J 2013; 42: 1503-1512
Year: 2013



Monitoring asthma in children: lung function, bronchial hyperresponsiveness and inflammation
Source: International Congress 2014 – Monitoring asthma in children: results of the ERS Task Force
Year: 2014



Relationship of atopy, asthma severity and bronchial hyperresponsiveness
Source: Eur Respir J 2001; 18: Suppl. 33, 429s
Year: 2001

The influence of smoking on the formation of cold airway hyperresponsiveness and destructive-cytolytic profile of goblet cells in asthma patients
Source: International Congress 2018 – Studying novel biomarkers in asthma and COPD
Year: 2018


Effects of smoking and atopy on bronchial hyperresponsiveness (BHR)
Source: Eur Respir J 2003; 22: Suppl. 45, 415s
Year: 2003

Different associations of inflammation and current smoking with bronchial hyperresponsiveness (BHR) to histamine and BHR to AMP in families with asthma
Source: Eur Respir J 2002; 20: Suppl. 38, 410s
Year: 2002

ADAM33 is involved in post-natal airway hyperresponsiveness caused by maternal allergic airway inflammation
Source: International Congress 2018 – Paediatric asthma: new mechanisms and tools
Year: 2018


Galectn-3 : marker of airway inflammation in bronchial asthma
Source: International Congress 2018 – Studying novel biomarkers in asthma and COPD
Year: 2018


Osteopontin exacerbates airway hyperresponsiveness and lung remodeling in chronic asthma
Source: Annual Congress 2008 - Basic science in asthma and COPD
Year: 2008

Determinants of airway responsiveness to histamine in children
Source: Eur Respir J 2005; 25: 462-467
Year: 2005



The burden of airway hyperresponsiveness on the control of asthma
Source: Annual Congress 2011 - Update on monitoring airway diseases
Year: 2011


Adiposity, asthma and airway inflammation
Source: Eur Respir J 2006; 28: Suppl. 50, 235s
Year: 2006