e-learning
resources
Vienna 2012
Sunday, 02.09.2012
Cell biology and inflammatory gene expression in chronic lung disease
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Corticosteroid treatment selectively decreases mast cells in the smooth muscle and epithelium of asthmatic bronchi
A. James, P. Gyllfors, E. Henriksson, S.E. Dahlén, M. Adner, G. Nilsson, B. Dahlén (Stockholm, Huddinge, Sweden)
Source:
Annual Congress 2012 - Cell biology and inflammatory gene expression in chronic lung disease
Session:
Cell biology and inflammatory gene expression in chronic lung disease
Session type:
Thematic Poster Session
Number:
804
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
A. James, P. Gyllfors, E. Henriksson, S.E. Dahlén, M. Adner, G. Nilsson, B. Dahlén (Stockholm, Huddinge, Sweden). Corticosteroid treatment selectively decreases mast cells in the smooth muscle and epithelium of asthmatic bronchi. Eur Respir J 2012; 40: Suppl. 56, 804
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
From pulmonary gas exchange to the interpretation of arterial blood gases
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Related content which might interest you:
Beta2-agonists but not glucocorticoids inhibit proliferation of asthmatic airway smooth muscle cells
Source: Eur Respir J 2004; 24: Suppl. 48, 251s
Year: 2004
Human lung mast cells adhere to airway smooth muscle
Source: Eur Respir J 2004; 24: Suppl. 48, 454s
Year: 2004
Eosinophils promote airway smooth muscle cells differentiation to the contractile phenotype in asthma
Source: International Congress 2019 – Novel mechanisms elucidated by translational studies of asthma
Year: 2019
A comparison of beta
2
-adrenoceptor desensitisation induced by olodaterol and formoterol in human lung mast cells and airway smooth muscle cells
Source: Annual Congress 2013 –New drugs in respiratory medicine
Year: 2013
Effect of p21Ras intervention on the phenotype of airway smooth muscle cells from asthmatic rats
Source: Annual Congress 2010 - Airway smooth muscle cells
Year: 2010
In vivo allergen-activated inflammatory eosinophils reduced airway smooth muscle cells apoptosis in allergic asthma patients
Source: Virtual Congress 2021 – New insights into the airway immunology of lung disease
Year: 2021
Co-culture of human airway epithelial cells alter the proliferation of human airway smooth muscle cells
Source: Eur Respir J 2002; 20: Suppl. 38, 572s
Year: 2002
Eosinophil subtypes’ effect on airway smooth muscle cells differentiation and migration in asthma
Source: Virtual Congress 2020 – Respiratory homeostasis and structural changes in disease
Year: 2020
Eosinophils promote airway smooth muscle cells contractility and a-actin gene expression in asthma
Source: International Congress 2018 – Identification of therapeutic targets for asthma
Year: 2018
Inhaled budesonide induces corticosteroid-dependent gene expression in asthmatics: Validation in primary epithelial and airways smooth muscle cells
Source: Annual Congress 2011 - Models of disease and drug actions
Year: 2011
Increased activation of TGF-β by asthmatic airway smooth muscle cells via integrin αVβ5
Source: Annual Congress 2009 - Airway cell biology
Year: 2009
Mitochondria alteration in airway smooth muscle cells from severe asthmatics
Source: Annual Congress 2008 - Mitochondria in muscle-related respiratory disorders
Year: 2008
Mast cell regulation of airway smooth muscle function in asthma
Source: Eur Respir J 2007; 29: 827-830
Year: 2007
Ultrastructure analysis of airway smooth muscle cells in fatal asthma
Source: Annual Congress 2008 - Basic science in asthma and COPD II
Year: 2008
Rolipram and salmeterol block β
1
/β
2
-integrin-mediated adhesion of eosinophils to cultured airway smooth muscle cells from non-asthmatic and asthmatic donors
Source: Eur Respir J 2003; 22: Suppl. 45, 454s
Year: 2003
Decreased hyaluronan in airway smooth muscle cells of asthma and COPD patients
Source: Annual Congress 2009 - Hot topics in molecular pathology and functional genomics of COPD
Year: 2009
Inhibition of IgE-induced airway smooth muscle cell constriction by the anti-IgE-antibody omalizumab
Source: Annual Congress 2008 - Airway cell biology and immunology
Year: 2008
The effect of gamma-tocotrienol on human airway smooth muscle cell proliferation and migration
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013
Role of bronchial smooth muscle mitochondria in airway remodeling in asthma
Source: Eur Respir J 2007; 30: Suppl. 51, 286s
Year: 2007
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept