Investigating the role of IREB2 genetic variants in susceptibility to COPD

N. Kalsheker, S. Chappell, T. Guetta-Baranes, A. Alsaegh (United Kingdom; Makkah, Saudi Arabia)

Source: Annual Congress 2012 - Gene-environment treatment and asthma
Session: Gene-environment treatment and asthma
Session type: Poster Discussion
Number: 472
Disease area: Airway diseases

Congress or journal article abstractE-poster

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
N. Kalsheker, S. Chappell, T. Guetta-Baranes, A. Alsaegh (United Kingdom; Makkah, Saudi Arabia). Investigating the role of IREB2 genetic variants in susceptibility to COPD. Eur Respir J 2012; 40: Suppl. 56, 472

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
The role of LTA4H and ALOX5AP polymorphisms in asthma susceptibility
Source: Eur Respir J 2007; 30: Suppl. 51, 130s
Year: 2007

Positive association of PPARG2 and PPARGC1A gene polymorphisms with sarcoidosis susceptibility
Source: Annual Congress 2007 - Novel therapeutic targets and disease markers in pulmonary pathology
Year: 2007



Role of AQP5 polymorphisms in predisposition to asthma
Source: International Congress 2019 – Novel methods, diagnostics and biomarkers in chronic lung diseases
Year: 2019


The role of ADAM33 polymorphisms in two German populations
Source: Eur Respir J 2006; 28: Suppl. 50, 251s
Year: 2006

Presence of Cox2 polymorphisms and susceptibility to sarcoidosis and its evolution
Source: Eur Respir J 2007; 30: Suppl. 51, 113s
Year: 2007

Genetic determinants of lung function in COPD: A genome-wide association study
Source: Annual Congress 2010 - Genetics and risk factors for asthma and COPD
Year: 2010

Identification of novel rare genetic variants associated with COPD in the general population
Source: International Congress 2018 – Multiomics studies in epidemiology: what can they tell us?
Year: 2018



A BTNL2 gene variant confers to sarcoidosis susceptibility by an increased risk towards the chronic form of the disease
Source: Eur Respir J 2006; 28: Suppl. 50, 148s
Year: 2006

Relation of genetic polymorphisms inflammatory pathway genes with occupational chronic bronchitis susceptibility
Source: Annual Congress 2010 - Chemical exposures and respiratory effects
Year: 2010


M2 allele of alpha1-antitrypsin gene and genetic susceptibility to COPD in Korean men
Source: Eur Respir J 2005; 26: Suppl. 49, 442s
Year: 2005

Associations between genetic polymorphisms and drug response phenotypes in patients with asthma
Source: Eur Respir J 2007; 30: Suppl. 51, 360s
Year: 2007

The role of 774C/T, -691C/T and 894G/T polymorphisms of NOS3 gene in development of COPD
Source: Eur Respir J 2004; 24: Suppl. 48, 316s
Year: 2004

Genetic polymorphisms and lung cancer susceptibility in a Brazilian population
Source: Annual Congress 2008 - Advances in functional genomics of neoplastic and non-neoplastic lung diseases
Year: 2008

The role of C/T polymorphism of NOS1 gene in COPD pathogenesis
Source: Eur Respir J 2005; 26: Suppl. 49, 96s
Year: 2005

The pharmacogenetic effect of ADRB2 polymorphisms on therapeutic response in COPD
Source: Annual Congress 2012 - COPD treatment and varia
Year: 2012


Rare genetic variants in genes with ciliary function underlie non-smoking related COPD
Source: International Congress 2017 – Rare and common genetic variants of common and rare airway diseases
Year: 2017

Human genetic variability and susceptibility to pulmonary TB
Source: Eur Respir Mon 2012; 58: 38-58
Year: 2012


A genome-wide association study on the risk variations for susceptibility to early COPD
Source: International Congress 2019 – New molecular signatures in COPD, lung cancer and a1-antitrypsin deficiency
Year: 2019


Investigating tensin1 expression and the presence of SNP identified in GWAS in COPD
Source: International Congress 2016 – Mechanisms of disease
Year: 2016