Airway smooth muscle-derived fractalkine needs VIP to induce mast cell chemotaxis

P. Berger, A. El-Shazly, P. O. Girodet, R. Marthan, M. Tunon de Lara (Bordeaux, France)

Source: Annual Congress 2005 - Airway regulation and response II
Session: Airway regulation and response II
Session type: Thematic Poster Session
Number: 870
Disease area: Airway diseases

Congress or journal article abstract

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
P. Berger, A. El-Shazly, P. O. Girodet, R. Marthan, M. Tunon de Lara (Bordeaux, France). Airway smooth muscle-derived fractalkine needs VIP to induce mast cell chemotaxis. Eur Respir J 2005; 26: Suppl. 49, 870

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Eotaxin/CCL11 inhibits proliferation of human airway smooth muscle cells
Source: Eur Respir J 2005; 26: Suppl. 49, 127s
Year: 2005

Asthmatic and non-asthmatic airway smooth muscle production of the mast cell chemotaxin CXCL10 involves MAPK
Source: Eur Respir J 2006; 28: Suppl. 50, 854s
Year: 2006

Human airway smooth muscle cell and mast cell interaction is mediated via CD44 and CD51 in asthma
Source: Eur Respir J 2005; 26: Suppl. 49, 712s
Year: 2005

Omalizumab inhibits IgE-induced cytokine secretion by human airway smooth muscle cells
Source: Annual Congress 2008 - Basic science in asthma and COPD
Year: 2008

LSC Abstract – Eosinophils enhance WNT-5a and TGF-β1 genes expression in airway smooth muscle cells and promote their proliferation in asthma
Source: International Congress 2016 – Clinical characteristics of asthma
Year: 2016

Eosinophil subtypes’ effect on airway smooth muscle cells differentiation and migration in asthma
Source: Virtual Congress 2020 – Respiratory homeostasis and structural changes in disease
Year: 2020


Eotaxin and CCR3 expression in cultured human airway smooth muscle cells
Source: Eur Respir J 2002; 20: Suppl. 38, 2s
Year: 2002

MNK-1 inhibition reduces proliferation and CXCL10 in airway smooth muscle cells
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013

Mitogenic effects of RBL-1 cell mediators on human airway smooth muscle (ASM) cells are via nonproteolitic actions
Source: Eur Respir J 2003; 22: Suppl. 45, 81s
Year: 2003

Human lung mast cells adhere to airway smooth muscle
Source: Eur Respir J 2004; 24: Suppl. 48, 454s
Year: 2004

IL-4/IL-13 promote Th2-type inflammatory changes in airway smooth muscle and induce myofibroblast differentiation
Source: Eur Respir J 2005; 26: Suppl. 49, 224s
Year: 2005

IgE-induced pro-inflammatory extracellular matrix composition in airway smooth muscle cells is prevented by omalizumab
Source: Annual Congress 2012 - The best of pharmacology treatment for asthma and COPD
Year: 2012


Tocotrienol inhibits TGF-beta1-induced human airway smooth muscle cell differentiation
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013


Stimulation of pulmonary smooth muscle cell proliferation by endothelial progenitor cells
Source: Annual Congress 2008 - Vascular and interstitial remodelling in chronic lung disease: therapeutic interventions on the horizon
Year: 2008

Integrins regulate eosinophil adhesion to the airway smooth muscle cells in asthma patients
Source: International Congress 2015 – Asthma mechanisms and management
Year: 2015

Cysteinyl leukotrienes augment human airway smooth muscle migration
Source: Eur Respir J 2002; 20: Suppl. 38, 384s
Year: 2002

Co-culture of human airway epithelial cells alter the proliferation of human airway smooth muscle cells
Source: Eur Respir J 2002; 20: Suppl. 38, 572s
Year: 2002

The effect of gamma-tocotrienol on human airway smooth muscle cell proliferation and migration
Source: Annual Congress 2013 –Airway smooth muscle and cell biology
Year: 2013


Eosinophils promote airway smooth muscle cells differentiation to the contractile phenotype in asthma
Source: International Congress 2019 – Novel mechanisms elucidated by translational studies of asthma
Year: 2019