e-learning
resources
Amsterdam 2011
Monday, 26.09.2011
Treatment options and tumour biology of malignant pleural mesothelioma
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
The regulatory effect of microRNAs on STAT signaling in malignant mesothelioma
L. Arzt, H. Kothmaier, F. Quehenberger, I. Halbwedl, H. H. Popper (Graz, Austria)
Source:
Annual Congress 2011 - Treatment options and tumour biology of malignant pleural mesothelioma
Session:
Treatment options and tumour biology of malignant pleural mesothelioma
Session type:
Oral Presentation
Number:
2949
Disease area:
Thoracic oncology
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
L. Arzt, H. Kothmaier, F. Quehenberger, I. Halbwedl, H. H. Popper (Graz, Austria). The regulatory effect of microRNAs on STAT signaling in malignant mesothelioma. Eur Respir J 2011; 38: Suppl. 55, 2949
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Impact of Dexamethasone on pathogen profile of COVID-19 patients requiring intensive care: a multicentre retrospective study
Muscle energy techniques for COPD patients: Effects on pulmonary function and activities of daily living
Related content which might interest you:
Dissecting the mTOR pathway in malignant pleural mesothelioma
Source: Annual Congress 2010 - Basic mechanisms in malignant pleural disease biology
Year: 2010
Epigenetic deregulated translation control of C/EBP-alpha leads to increased mesothelioma cell proliferation
Source: Annual Congress 2011 - Treatment options and tumour biology of malignant pleural mesothelioma
Year: 2011
LSC - 2017 - Targeting CD98hc: a key mediator of tumour and microenvironmental crosstalk in malignant mesothelioma
Source: International Congress 2017 – Mesothelioma and malignant pleural disease
Year: 2017
Proliferative intercellular signalling pathway in human malignant mesothelioma cells
Source: Annual Congress 2010 - Basic mechanisms in malignant pleural disease biology
Year: 2010
CREB signaling in KRAS-driven lung adenocarcinoma
Source: ERS Lung Science Conference 2017
Year: 2017
Epigenetic deregulated translation control of C/EBP-alpha leads to increased mesothelioma cell proliferation
Source: Annual Congress 2011 - Pleural and mediastinal malignancies: management and rare clinical cases
Year: 2011
Micro RNA-21 Targeting PI3K/PTEN/Akt signaling axis regulates multiple biological functions in human lung cancer
Source: International Congress 2018 – Lung cancer: from the bench to the bedside
Year: 2018
BMP-Smad1 target gene Wnt inhibitory factor 1 (WIF1) coordinates BMP and Wnt signaling activities in regulating lung development in vivo
Source: Annual Congress 2010 - New mechanisms in lung development, acute and chronic lung diseases
Year: 2010
HMGA2 mediated epigenetic regulation of Gata6 controls epithelial WNT signaling during lung development
Source: International Congress 2017 – Molecular biology of normal and abnormal lung development
Year: 2017
Hedgehog signalling crosstalks orchestrate human lung development
Source: Virtual Congress 2020 – Cellular stress in respiratory diseases
Year: 2020
Effects of mTOR Inhibition in malignant mesothelioma
Source: Annual Congress 2010 - Basic mechanisms in malignant pleural disease biology
Year: 2010
Wnt and TGF-beta signaling in lung development
Source: Lung Science Conference 2009 - Cell proliferation, Differentiation and Carcinogenesis
Year: 2009
Global miRNA expression in Treg cells from patients with pulmonary sarcoidosis indicates for targeting of MAPK and FOXO signaling pathways
Source: International Congress 2015 – Latest news on miRNAs in pulmonary research
Year: 2015
The Hippo pathway effectors TAZ and YAP are sequentially required in lung development.
Source: International Congress 2019 – Molecular and experimental paediatric lung disease
Year: 2019
Roles of Rho-Rock and Smad pathways in TGFβ1-mediated lung fibroblast differentiation
Source: Annual Congress 2008 - Regulatory mechanisms of cell activation, renewal and recruitment
Year: 2008
Phagosome-regulated mTOR signalling during sarcoidosis granuloma biogenesis
Source: Eur Respir J, 57 (3) 2002695; 10.1183/13993003.02695-2020
Year: 2021
CXCL12 expression in malignant mesothelioma
Source: Eur Respir J 2007; 30: Suppl. 51, 138s
Year: 2007
mTOR inhibition blocks tumor growth and pleural fluid accumulation in experimental murine mesothelioma
Source: Annual Congress 2011 - Treatment options and tumour biology of malignant pleural mesothelioma
Year: 2011
NTHi-promoted proliferation of K-ras-induced early lung cancer lesions is fully dependent on Toll-like receptor signaling
Source: International Congress 2017 – Basic research in lung cancer: a scientific potpourri from genetic alterations to immune cells
Year: 2017
PP222 – Forkhead Box F1 as a mediator of Wnt signaling in human lung endothelial cells
Source: ERS Lung Science Conference 2021
Year: 2021
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept