e-learning
resources
Amsterdam 2011
Sunday, 25.09.2011
Experimental models and research in diffuse parenchymal lung diseases
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Modulation of fibroblast phenotype in idiopathic pulmonary fibrosis: Role of Nrf2
E. Artaud-Macari, D. Goven, S. Brayer, J. Marchal-Sommé, B. Crestani, A. Boutten, M. Bonay (Paris, France)
Source:
Annual Congress 2011 - Experimental models and research in diffuse parenchymal lung diseases
Session:
Experimental models and research in diffuse parenchymal lung diseases
Session type:
Thematic Poster Session
Number:
680
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
E. Artaud-Macari, D. Goven, S. Brayer, J. Marchal-Sommé, B. Crestani, A. Boutten, M. Bonay (Paris, France). Modulation of fibroblast phenotype in idiopathic pulmonary fibrosis: Role of Nrf2. Eur Respir J 2011; 38: Suppl. 55, 680
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: ANCA associated vasculitis and lung disease
Panel discussion on genetics in pulmonary fibrosis
From pulmonary gas exchange to the interpretation of arterial blood gases
Related content which might interest you:
Reactivation of developmental pathways in idiopathic pulmonary fibrosis: FGF9 and FGF18 modulate the phenotype of control and fibrotic human lung fibroblasts in vitro
Source: International Congress 2015 – IPF: from bench to bedside
Year: 2015
Role of Bcl-xL in hepatocyte growth factor elicited epithelial protection in idiopathic lung fibrosis
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis: pathogenesis
Year: 2013
NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFβ1-induced fibroblast differentiation into myofibroblasts
Source: Annual Congress 2009 - From emphysema to fibrosis: mechanisms of cellular remodelling in the diseased lung
Year: 2009
Expression of macrophage migration inhibitory factor in patients with idiopathic pulmonary fibrosis
Source: International Congress 2015 – Interstitial lung diseases II
Year: 2015
Mast cells and fibroblasts work in concert to aggravate pulmonary fibrosis: Role of transmembrane SCF and PAR-2/PKCalpha/Raf-1/p44/42 signaling pathway
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
Dysregulation of the TGF-beta system in idiopathic pulmonary fibrosis
Source: Eur Respir J 2004; 24: Suppl. 48, 10s
Year: 2004
Fibroblast growth factor 9 (FGF9) modulates the phenotype of control and fibrotic human lung fibroblasts
in vitro
Source: International Congress 2014 – New and old players in fibrotic lung disease
Year: 2014
Effect of pirfenidone on TGF-beta-induced pro-fibrotic effects in primary human lung cells derived from patients with idiopathic pulmonary fibrosis
Source: International Congress 2014 – ILDs 2
Year: 2014
Uncoupling the pro-fibrotic effects of TGF-beta1 versus tissue hardening in lung fibroblasts
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
The role of epithelium-dependent fibroblast activation in pulmonary fibrosis
Source: Virtual Congress 2021 – Chinese programme 2021
Year: 2021
Bromodomain inhibitor JQ1 attenuates TGF-beta-suppressed
SOD2
expression and reduces proliferation in human primary pulmonary fibroblasts
Source: Annual Congress 2013 –The multiple faces of stress in lung injury
Year: 2013
Role of MUC1 in idiopathic pulmonary fibrosis
Source: International Congress 2016 – ILD pathogenesis
Year: 2016
Tranilast inhibits pulmonary fibrosis by suppressing TGFbeta-mediated extracellular matrix protein production
Source: Annual Congress 2013 –Diffuse pulmonary fibrosis
Year: 2013
Role of IL-11 in vascular function of pulmonary fibrosis patients
Source: International Congress 2019 – Pulmonary hypertension with lung diseases and experimental pulmonary hypertension
Year: 2019
Role of the inflammasome in idiopathic pulmonary fibrosis
Source: International Congress 2017 – New insights into pathogenesis of lung disease
Year: 2017
Does apoptosis evasion only affect fibroblasts from idiopathic pulmonary fibrosis?
Source: Annual Congress 2013 –Cell signalling and inflammation: what's new in 2013?
Year: 2013
Epidermal growth factor receptor (EGFR) activation is required for TGFbeta1-induced epithelial-mesenchymal transition (EMT) in idiopathic pulmonary fibrosis (IPF)
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Mesenchyme associated transcription factor PRRX1: A key regulator of IPF fibroblast
Source: International Congress 2016 – Repair pathways: from lung development to ageing of the lung
Year: 2016
Role of MUC4 in idiopathic pulmonary fibrosis
Source: International Congress 2018 – Back to basics and translational research in idiopathic interstitial pneumonias
Year: 2018
Role of MUC4 in idiopathic pulmonary fibrosis
Source: International Congress 2019 – Translational aspects of idiopathic pulmonary fibrosis
Year: 2019
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept