Reciprocal regulation of iNOS and PARP-1 during allergen-induced eosinophilia

Naura A. S., Datta R., Hans C. P., Zerfaoui M., Rezk B. M., Errami Y., Oumouna M., Matrougui K., Boulares A. H.

Source: Eur Respir J 2009; 33: 252-262
Journal Issue: February
Disease area: Airway diseases

Congress or journal article abstractFull text journal articlePDF journal article, handout or slides

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
Naura A. S., Datta R., Hans C. P., Zerfaoui M., Rezk B. M., Errami Y., Oumouna M., Matrougui K., Boulares A. H.. Reciprocal regulation of iNOS and PARP-1 during allergen-induced eosinophilia. Eur Respir J 2009; 33: 252-262

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Local application of FTY720 to the lung abrogates experimental asthma by altering myeloid dendritic cell function
Source: Annual Congress 2006 - Towards understanding the pathogenesis of asthma
Year: 2006


IL-37 requires IL18Rα and SIGIRR to diminish experimental allergic airway inflammation in mice
Source: International Congress 2015 – Animal studies of asthma and other inflammatory conditions
Year: 2015

Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice
Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma
Year: 2007



CCL6 produced by eosinophils drives allergic airway inflammation in mice
Source: International Congress 2019 – Modelling and monitoring of airway diseases
Year: 2019

Role of macrophage migration inhibitory factor in ovalbumin-induced airway inflammation in rats
Source: Eur Respir J 2006; 27: 726-734
Year: 2006



IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide- but not allergen-induced airway neutrophilia
Source: Eur Respir J 2002; 20: Suppl. 38, 618s
Year: 2002

Inhibitors of PDE4 delay eosinophil apoptosis
Source: Annual Congress 2005 - Improving asthma control in the harder-to-reach population
Year: 2005


Differential gene expression and cytokine production from neutrophils in asthma phenotypes
Source: Eur Respir J 2010; 35: 522-531
Year: 2010



Overproduction of mature IL-18 induces IL-13 producing CD4+Tcells in the lungs, and results in airway hyperresponsiveness and pulmonary inflammation in Balb/c mice
Source: Annual Congress 2010 - Asthma: basic science and clinical studies
Year: 2010

Pathology, inflammation and cytokines of severe asthma
Source: Eur Respir Mon 2011; 51: 97-106
Year: 2011


Inflammatory cell activation in eosinophilic and non-eosinophilic asthma
Source: Annual Congress 2006 - Immunology of the airways
Year: 2006


ST2 gene knockout attenuates allergic airways disease
Source: Annual Congress 2006 - Immunological mechanisms in cell recruitment to the lungs
Year: 2006


Increase in laminin expression in allergic airway remodelling and decrease by dexamethasone
Source: Eur Respir J 2004; 24: 107-115
Year: 2004



Role of Rho kinase isoforms in murine allergic airway responses
Source: Eur Respir J 2011; 38: 841-850
Year: 2011



Inhaled doxycycline reduces allergen-induced airway inflammation, hyperresponsiveness and remodelling in mice
Source: Annual Congress 2005 - Inflammation and remodelling in allergy and asthma
Year: 2005


Upregulated response to chemokines in oxidative metabolism of eosinophils in asthma and allergic rhinitis
Source: Eur Respir J 2003; 21: 925-931
Year: 2003



High-level expression of PD-L1 by DC suppresses airway inflammation in OVA-induced allergic asthma
Source: Annual Congress 2010 - Animal models of asthma and lung inflammation
Year: 2010

Modulation of ozone-induced airway hyperresponsiveness and inflammation by interleukin-13
Source: Eur Respir J 2008; 32: 571-578
Year: 2008



Schistosome-induced pulmonary B cells inhibit allergic airway inflammation and display a reduced Th2-driving function
Source: International Congress 2015 – Animal studies of asthma and other inflammatory conditions
Year: 2015

Dissociation of airway inflammation and hyperresponsiveness by cyclooxygenase inhibition in allergen challenged mice
Source: Eur Respir J 2009; 34: 200-208
Year: 2009