In vivo blockade of CTLA-4 counteracts inhalation tolerance and leads to development of allergic airway inflammation in mice repeatedly exposed to aerosolized ovalbumin Source: Eur Respir J 2004; 24: Suppl. 48, 6s Year: 2004
Evidence for secretion of IL-18 in asthmatic airway inflammation assessed by endobronchial allergen challenge and its relationship to accumulation of inflammatory cells Source: Eur Respir J 2001; 18: Suppl. 33, 531s Year: 2001
VIP knockout mice show asthma-like airway hyper-reactivity, and inflammatory response to immunologic challenge Source: Eur Respir J 2004; 24: Suppl. 48, 658s Year: 2004
T-cell mediated inflammation is is not required for sustained AHR that persists in mice following chronic allergen challenge Source: Eur Respir J 2003; 22: Suppl. 45, 482s Year: 2003
The development of an airway hyperresponsiveness (AHR) depends on mast cells following sensitization without adjuvant Source: Annual Congress 2006 - Cellular immunology in asthma pathogenesis Year: 2006
Effects of OVA-induced allergic airway inflammation and remodeling in mice in three distinct asthma models Source: Annual Congress 2010 - Animal models of asthma and lung inflammation Year: 2010
The airway inflammatory response in allergic mice is downregulated by budesonide Source: Eur Respir J 2001; 18: Suppl. 33, 265s Year: 2001
Airway allergy in pregnant mice: reduced Th2 responses but enhanced airway hyperresponsiveness Source: Eur Respir J 2006; 28: Suppl. 50, 590s Year: 2006
Deficiency of MMP-19 promotes allergen-induced eosinophil burden and airway responsiveness in mice Source: Annual Congress 2007 - Regulation of allergic airway inflammation in animal models of asthma Year: 2007
Effects of active and passive smoking on allergic airway inflammation and airway hyperresponsiveness in mice Source: Eur Respir J 2002; 20: Suppl. 38, 310s Year: 2002
Interferon-γ-pretreated dendritic cells induced dual Th1 and Th2 responses and failed to prevent development of allergic airway inflammation Source: Eur Respir J 2006; 28: Suppl. 50, 230s Year: 2006
CCL6 produced by eosinophils drives allergic airway inflammation in mice Source: International Congress 2019 – Modelling and monitoring of airway diseases Year: 2019
Airway remodelling and hyperresponsiveness are enhanced in immature vs adult mice after active allergenic sensitization Source: Annual Congress 2008 - Assessment of inflammation, hyperresponsiveness and response to exercise in asthmatic children Year: 2008
Moderate aerobic conditioning decreases allergic airway inflammation and airway remodeling in chronically sensitized mice Source: Annual Congress 2006 - Immunology of the airways Year: 2006
Intranasal dust exposure decreases Th2 driven allergic inflammation and increases the Th17 response in mice Source: Annual Congress 2009 - Animal models of airways inflammation Year: 2009
Different effects of cox-inhibition on airway inflammation and hyperresponsiveness in mice Source: Eur Respir J 2007; 30: Suppl. 51, 713s Year: 2007
Effects of chronic allergen exposure on the airway hyperreactivity in sensitized rats Source: Annual Congress 2011 - Animal models of airway inflammation Year: 2011
Transgenic mice overexpressing IL-18 display increased IL-13 in the airways but no changes of the airway hyperresponsiveness in a murine model of late-phase asthma: evidence for a dual role of IL-18 on mucosal Th1/Th2 responses Source: Eur Respir J 2002; 20: Suppl. 38, 1s Year: 2002
TNF-α negatively modulates allergen-induced airway hyperresponsiveness through γδ T cells Source: Eur Respir J 2002; 20: Suppl. 38, 27s Year: 2002
Effect of previous physical conditioning on pulmonary inflammation and allergic immune response in a chronic allergic airway inflammation in mice Source: Eur Respir J 2007; 30: Suppl. 51, 370s Year: 2007