e-learning
resources
ERJ
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Mitochondrial antiviral signaling protein is crucial for the development of pulmonary fibrosis
Sang-Hun Kim, Jung Yeon Lee, Chang Min Yoon, Hyeon Jun Shin, Sei Won Lee, Ivan Rosas, Erica Herzog, Charles S. Dela Cruz, Naftali Kaminski, Min-Jong Kang
Source:
Eur Respir J, 57 (4) 2000652; 10.1183/13993003.00652-2020
Journal Issue:
April
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Sang-Hun Kim, Jung Yeon Lee, Chang Min Yoon, Hyeon Jun Shin, Sei Won Lee, Ivan Rosas, Erica Herzog, Charles S. Dela Cruz, Naftali Kaminski, Min-Jong Kang. Mitochondrial antiviral signaling protein is crucial for the development of pulmonary fibrosis. Eur Respir J, 57 (4) 2000652; 10.1183/13993003.00652-2020
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: Diagnostic tools for Obstructive Sleep Apnoea in adults and children
Expert interview: Physiological classification of lung function impairment
Occupational exposures to respiratory diseases: A case-based discussion
Related content which might interest you:
MAV(S)erick mitochondria: an unconventional role for mitochondrial antiviral signalling protein in pulmonary fibrosis
Source: Eur Respir J, 57 (4) 2004500; 10.1183/13993003.04500-2020
Year: 2021
mTOR signalling is an essential pathway for TGF-β1 induced collagen synthesis
Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease
Year: 2015
ELMOD2, a candidate gene for idiopathic pulmonary fibrosis, regulates antiviral responses
Source: Annual Congress 2010 - Lung cell biology and immunology
Year: 2010
Extracellular HSP90a promotes cellular senescence in pulmonary fibrosis through TGFß signaling
Source: Virtual Congress 2020 – Idiopathic pulmonary fibrosis and interstitial lung disease: progression, genes and more
Year: 2020
PP208 – SRC family kinases modulate molecular pathways associated with mitochondrial dysfunction in idiopathic pulmonary fibrosis
Source: ERS Lung Science Conference 2021
Year: 2021
Defective p53-mediated pro-apoptotic pathway in idiopathic pulmonary fibrosis
Source: Annual Congress 2008 - New insights into the pathogenesis of idiopathic pulmonary fibrosis and sarcoidosis
Year: 2008
TRIM33 prevents pulmonary fibrosis by impairing TGF-ß1 signalling
Source: Eur Respir J, 55 (6) 1901346; 10.1183/13993003.01346-2019
Year: 2020
PGAM5 acts as a key mediator of lung fibrosis by disrupting mitochondrial homeostasis
Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease
Year: 2019
The potential role of antimicrobial peptides from wasp as regulators of the fibrotic process in idiopathic pulmonary fibrosis.
Source: International Congress 2019 – Assessment and management of immune-mediated interstitial lung diseases
Year: 2019
The matrix-fibroblast interplay contributes to HIF-1 alpha pathway activation: a possible therapeutic target in idiopathic pulmonary fibrosis (IPF)
Source: Virtual Congress 2020 – Elucidating the mechanisms underlying lung repair and exploring therapeutic strategies in vitro
Year: 2020
The pathomechanistic role of autophagy in lung fibrosis
Source: International Congress 2017 – Interstitial lung diseases (ILDs): novel pathogenetic pathways
Year: 2017
Mast cells and fibroblasts work in concert to aggravate pulmonary fibrosis: Role of transmembrane SCF and PAR-2/PKCalpha/Raf-1/p44/42 signaling pathway
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
SH2 domain-containing phosphatase-SHP-2 is a novel anti-fibrotic regulator in pulmonary fibrosis
Source: International Congress 2016 – Emerging concepts in lung disease pathogenesis
Year: 2016
Regulation of alternative macrophage activation by galectin-3; potential role in lung fibrosis
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009
The role of extracellular vesicles in phenotypic alteration of lung cells in pulmonary fibrosis
Source: Annual Congress 2013 –The role of hormones and cytokines in lung injury
Year: 2013
Chaotic activation of developmental signalling pathways drives idiopathic pulmonary fibrosis
Source: Eur Respir Rev, 29 (158) 190140; 10.1183/16000617.0140-2019
Year: 2020
Tranilast inhibits pulmonary fibrosis by suppressing TGFbeta-mediated extracellular matrix protein production
Source: Annual Congress 2013 –Diffuse pulmonary fibrosis
Year: 2013
Micro-RNA-125a/b target A20 and MAVS to promote inflammatory and impair antiviral responses in chronic obstructive pulmonary disease
Source: International Congress 2016 – Mechanisms of disease
Year: 2016
PP121 – Gaq/11 and Ga12/13 promote pulmonary fibrosis by driving distinct myofibroblast functions
Source: ERS Lung Science Conference 2021
Year: 2021
Reactivation of developmental pathways in idiopathic pulmonary fibrosis: FGF9 and FGF18 modulate the phenotype of control and fibrotic human lung fibroblasts in vitro
Source: International Congress 2015 – IPF: from bench to bedside
Year: 2015
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept