Source: Annual Congress 2007 - Acute lung injury and inflammation
Disease area: Respiratory critical care



Rating:

Share or cite this content

Citations should be made in the following way:
F. Arbol, C. Casadevall, V. Merlo, E. Sánchez, J. Gea (Talavera, Castilla-La Mancha, Barcelona, Catalonia, Spain). Sepsis induces overexpression of proinflammatory myokines in the intercostal muscle. Eur Respir J 2007; 30: Suppl. 51, 2635

Member's Comments

No comment yet.

Related content which might interest you: Upregulation of proinflammatory cytokine genes in the external intercostal muscle of COPD patients Source: Eur Respir J 2005; 26: Suppl. 49, 362s Year: 2005 Upregulation of pro-inflammatory cytokines in the intercostal muscles of COPD patients Source: Eur Respir J 2007; 30: 701-707 Year: 2007 Differential regulation of neurotrophin expression in human airway smooth muscle cells by pro-inflammatory cytokines Source: Eur Respir J 2002; 20: Suppl. 38, 383s Year: 2002 NOD1 and TLR4 synergise to elevate release of the pro-inflammatory cytokine CXCL8 in human pulmonary artery smooth muscle cells Source: Annual Congress 2010 - Sepsis and multiorgan failure Year: 2010 Heme-oxygenase (HO)-1 inhibits TNF-α induced CXCL10 secretion by airway smooth muscle cells Source: Annual Congress 2011 - Airway smooth muscle cells and fibroblasts: cell biology Year: 2011 Hypoxia sensitizes skeletal muscle to fasting-induced muscle atrophy; role of AMPK activation Source: International Congress 2015 – New insights into exercise and muscle performance Year: 2015 TNF-α-induced NF-κB activation stimulates skeletal muscle glycolytic metabolism through activation of HIF-1α Source: International Congress 2015 – New insights into lung disease pathogenesis Year: 2015 Plasminogen-stimulated inflammatory cytokine production by airway smooth muscle cells is regulated by annexin A2 Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Rhinovirus-induced IL-33 expression in asthmatic airway smooth muscle cells is TLR3-dependent and involves activation of TAK1 Source: International Congress 2016 – Emerging concepts in lung disease pathogenesis Year: 2016 Remicade suppresses cytokine-induced up-regulation of bradykinin receptor-mediated airway smooth muscle contractions Source: Annual Congress 2005 - Inflammation and the airway smooth muscle: cellular and pharmacological interactions Year: 2005 Compound A modulates steroid insensitive chemokines in airway smooth muscle cells via IRF-1 dependent and independent pathways Source: International Congress 2015 – New targets and modalities for the treatment of asthma and COPD Year: 2015 Double-stranded RNA upregulates TLR3 and induces pro-inflammatory, and innate immune cytokine expression in human bronchial smooth muscle cells Source: Annual Congress 2010 - Airway smooth muscle cells Year: 2010 Atrophy and increased expression of MuRF1 and atrogin in skeletal muscle following acute pulmonary inflammation Source: Eur Respir J 2006; 28: Suppl. 50, 622s Year: 2006 Resveratrol impairs the release of steroid-resistant inflammatory cytokines from human airway smooth muscle cells in COPD Source: Annual Congress 2010 - Airway cells as targets for anti-inflammatory drugs Year: 2010 Impaired inhibitory action of corticosteroids on chemokine expression induced by TNFalpha in airway smooth muscle (ASM) cells from patients with severe asthma Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 Dimethylfumarate inhibits CXCL10 via haem oxygenase-1 in airway smooth muscle Source: Eur Respir J 2013; 41: 195-202 Year: 2013 Hypoxia-induced muscle atrophy and impaired regulation of protein turnover are partially dependent on muscle glucocorticoid receptor signaling Source: International Congress 2015 – Promising novel findings in translational pulmonary research Year: 2015 Glutathionylation of IκB-α inhibits NF-κB dependent chemokine secretion in airway smooth muscle cells Source: Annual Congress 2010 - Regulation of airway pharmacology Year: 2010 Resveratrol is superior to dexamethasone in suppressing cytokine release from human airway smooth muscle cells exposed to lipoteichoic acid in COPD Source: Annual Congress 2013 –Airway smooth muscle and cell biology Year: 2013 IL-4/IL-13 promote Th2-type inflammatory changes in airway smooth muscle and induce myofibroblast differentiation Source: Eur Respir J 2005; 26: Suppl. 49, 224s Year: 2005