Source: Eur Respir J 2014; 43: 464-473
Disease area: Airway diseases



Rating:

Share or cite this content

Citations should be made in the following way:
Firszt Rafael, Francisco Dave, Church Tony D., Thomas Joseph M., Ingram Jennifer L., Kraft Monica. Interleukin-13 induces collagen type-1 expression through matrix metalloproteinase-2 and transforming growth factor-?1 in airway fibroblasts in asthma. Eur Respir J 2014; 43: 464-473

Member's Comments

No comment yet.

Related content which might interest you: Vascular endothelial growth factor modulates matrix metalloproteinase-9 expression in asthma Source: Eur Respir J 2006; 28: Suppl. 50, 444s Year: 2006 VEGF induces ECM synthesis and fibroblast activity in human lung fibroblasts Source: International Congress 2017 – Towards better treatments for lung disease: experimental pharmacology Year: 2017 Effects of neurotrophins on matrix metalloproteinase production in human bronchial smooth muscle cells Source: Eur Respir J 2005; 26: Suppl. 49, 128s Year: 2005 Connective tissue growth factor and transforming growth factor β induces extracellular matrix in asthmatic airway smooth muscle Source: Eur Respir J 2005; 26: Suppl. 49, 712s Year: 2005 TGF-β upregulates matrix metalloproteinase secretion by bronchial epithelial cells derived from lung allografts Source: Eur Respir J 2004; 24: Suppl. 48, 180s Year: 2004 Increased matrix metalloproteinase-9 concentration and activity after stimulation with interleukin-17A in mouse airways Source: Annual Congress 2004 - Insights into asthma and COPD Year: 2004 Secretion of wound-induced TGF-beta1 and TGF-beta2 enhance airway epithelial repair via HB-EGF and TGF-alpha Source: Annual Congress 2010 - Mechanisms and modulation of allergic inflammation in the lung Year: 2010 Matrix metalloproteinase (MMP)-2 production by fibroblasts and mast cells: the role in pulmonary fibrosis Source: Eur Respir J 2004; 24: Suppl. 48, 455s Year: 2004 Matrix metalloproteinase inhibitor regulates inflammatory cells migration through reducing ICAM-1 and VCAM-1 expression in a murine model of toluene diisocyanate-induced asthma Source: Eur Respir J 2003; 22: Suppl. 45, 483s Year: 2003 Transforming growth factor-β induces collagen synthesis without inducing IL-8 production in mesothelial cells Source: Eur Respir J 2003; 22: 197-202 Year: 2003 Regulation of matrix metalloproteinases and tissue inhibitors of matrix metalloproteinases expression in the bronchial secretions of asthmatics and COPD Source: Eur Respir J 2002; 20: Suppl. 38, 92s Year: 2002 CCL5 (RANTES) inhibits TGF-β1-induced collagen production in human bronchial epithelial cells by action of Smad protein Source: Annual Congress 2009 - New results in molecular pathology and functional genomics of non-neoplastic lung diseases Year: 2009 Inhibition of TGF- β 1-induced extracellular matrix production in primary human pulmonary fibroblasts by rapamycin Source: Annual Congress 2011 - Genetic and molecular background in pulmonary fibrosis Year: 2011 Tissue plasminogen activator decreased the expression of fibrogenic cytokines by doxycycline and transforming growth factor-beta in MeT-5A Source: Annual Congress 2010 - Current issues in malignant pleural diseases Year: 2010 Effects of keratinocyte growth factor (KGF) on matrix metalloproteinases (MMPs) & tissue inhibitors of metalloproteinases (TIMPs) mRNA levels in emphysematous rats lungs Source: Annual Congress 2008 - Inflammatory trigger mechanisms in asthma and COPD Year: 2008 Statins inhibit matrix metalloproteinase release from human lung fibroblasts Source: Eur Respir J 2010; 35: 637-646 Year: 2010 Discoidin domain receptor 1 regulates bronchial epithelial repair and matrix metalloproteinase production Source: Eur Respir J 2011; 37: 1482-1493 Year: 2011 In vitro profibrotic effects of rapamycin on basal or TGF-β1 induced primary human lung fibroblast: ECM production and turnover, migration and differentiation Source: Annual Congress 2012 - Diffuse parenchymal lung disease I Year: 2012 PP216 – Glucocorticoids exacerbate TGFß1 mediated fibrotic signalling in lung fibroblasts by inhibition of metalloproteinase expression and activity Source: ERS Lung Science Conference 2021 Year: 2021