e-learning
resources
Barcelona 2013
Wednesday, 11.09.2013
Growth factors at the crossroads of acute and chronic inflammation
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
A possible therapeutic agent for pulmonary fibrosis: Antibody against integrin alpha8beta1
N. Nishimichi, K. Kanno, K. Sentani, W. Yasui, Y. Yokosaki (Hiroshima, Japan)
Source:
Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Session:
Growth factors at the crossroads of acute and chronic inflammation
Session type:
Poster Discussion
Number:
4885
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
N. Nishimichi, K. Kanno, K. Sentani, W. Yasui, Y. Yokosaki (Hiroshima, Japan). A possible therapeutic agent for pulmonary fibrosis: Antibody against integrin alpha8beta1. Eur Respir J 2013; 42: Suppl. 57, 4885
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: ANCA associated vasculitis and lung disease
Panel discussion on genetics in pulmonary fibrosis
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Related content which might interest you:
A potential role for endothelial adhesion molecules and cytokines in chemotherapy-induced lung fibrosis
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Mast cells and fibroblasts work in concert to aggravate pulmonary fibrosis: Role of transmembrane SCF and PAR-2/PKCalpha/Raf-1/p44/42 signaling pathway
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
SH2 domain-containing phosphatase-SHP-2 is a novel anti-fibrotic regulator in pulmonary fibrosis
Source: International Congress 2016 – Emerging concepts in lung disease pathogenesis
Year: 2016
Metabolic shift during TGF- β-induced collagen synthesis
Source: International Congress 2016 – IPF pathogenesis
Year: 2016
Inhibition of collagen cross-links by novel LOXL2 selective inhibitors in an in-vitro model of fibroblastic foci of IPF
Source: International Congress 2016 – Common mechanisms in lung development and fibrosis
Year: 2016
Lipoxin (LXA
4
) as a novel therapy for idiopathic pulmonary fibrosis
Source: International Congress 2014 – New mechanisms in the pathogenesis of asthma and other lung diseases
Year: 2014
Romidepsin (FK228) target focal adhesion kinase (FAK) expression in lung fibrosis
Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease
Year: 2015
Uncoupling the pro-fibrotic effects of TGF-beta1 versus tissue hardening in lung fibroblasts
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
Does apoptosis evasion only affect fibroblasts from idiopathic pulmonary fibrosis?
Source: Annual Congress 2013 –Cell signalling and inflammation: what's new in 2013?
Year: 2013
The effects of tobramycin on pulmonary fibroblast function
Source: International Congress 2016 – Common mechanisms in lung development and fibrosis
Year: 2016
Proteasome function in lung fibrosis
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Pulmonary fibroblasts are affected by notch ligands in vitro
Source: International Congress 2015 – Airway remodelling: recent developments
Year: 2015
A role of Foxc2 gene in mice with bleomycin-induced pulmonary fibrosis
Source: International Congress 2015 – Pulmonary fibrosis: mechanisms of disease
Year: 2015
Severe lung inflammation is a feature of IPF
Source: Annual Congress 2013 –Idiopathic pulmonary fibrosis and rarities
Year: 2013
K
Ca
3.1 mediates alphaSMA expression in human lung myofibroblast via the Smad2/3 and TGFbeta1 signalling pathway
Source: International Congress 2014 – Cell biology 2014
Year: 2014
Increased fibrinolytic mediators in IPF as potential contributors to pulmonary fibrosis and vascular remodeling
Source: International Congress 2015 – IPF: from bench to bedside
Year: 2015
Bromodomain inhibitor JQ1 attenuates TGF-beta-suppressed
SOD2
expression and reduces proliferation in human primary pulmonary fibroblasts
Source: Annual Congress 2013 –The multiple faces of stress in lung injury
Year: 2013
Strong reversal of the lung fibrosis disease signature by autotaxin inhibitor GLPG1690 in a mouse model for IPF
Source: International Congress 2016 – Therapeutic horizons: novel targets and pharmacological models
Year: 2016
Effect of pirfenidone on TGF-beta-induced pro-fibrotic effects in primary human lung cells derived from patients with idiopathic pulmonary fibrosis
Source: International Congress 2014 – ILDs 2
Year: 2014
PAR-2 agonists induce TGF-beta and EMT leading to lung fibrosis
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept