e-learning
resources
ERJ
2010
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-?B and EGFR pathways
Hewson C.A., Haas J.J., Bartlett N.W., Message S.D., Laza-Stanca V., Kebadze T., Caramori G., Zhu J., Edbrooke M.R., Stanciu L.A., Kon O.M., Papi A., Jeffery P.K., Edwards M.R., Johnston S.L.
Source:
Eur Respir J 2010; 36: 1425-1435
Journal Issue:
December
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
Hewson C.A., Haas J.J., Bartlett N.W., Message S.D., Laza-Stanca V., Kebadze T., Caramori G., Zhu J., Edbrooke M.R., Stanciu L.A., Kon O.M., Papi A., Jeffery P.K., Edwards M.R., Johnston S.L.. Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-?B and EGFR pathways. Eur Respir J 2010; 36: 1425-1435
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Late Breaking Abstract - Implications of treatable traits and treatment choices on exacerbation risk in moderate-severe asthma
Management of Severe Asthma in Pediatric Patients by an Interdisciplinary Team in a Public Hospital Setting.
Respiratory sequelae of preterm birth across the life span
Related content which might interest you:
Expression of epidermal growth factor receptor (EGFR) in the bronchial epithelium of patients with chronic obstructive pulmonary disease (COPD)
Source: Annual Congress 2011 - COPD: clinical studies and animal cell models
Year: 2011
Altered expression of the Smad signalling pathway: implications for COPD pathogenesis
Source: Eur Respir J 2006; 28: 533-541
Year: 2006
Epidermal growth factor receptor-mediated innate immune responses and their roles in airway diseases
Source: Eur Respir J 2008; 32: 1068-1081
Year: 2008
The epidermal growth factor receptor mediates allergic airway remodelling in the rat
Source: Eur Respir J 2008; 32: 1213-1223
Year: 2008
Upregulation of basic fibroblast growth factor in smokers with chronic bronchitis
Source: Eur Respir J 2006; 27: 957-963
Year: 2006
The contribution of transforming growth factor-β and epidermal growth factor signalling to airway remodelling in chronic asthma
Source: Eur Respir J 2006; 27: 208-229
Year: 2006
Haemophilus influenzae
(NTHi) up-regulates acute inflammation in human lung tissue and causes downregulation of transforming growth factor-beta (TGF-beta) – A possible role of NTHi in remodeling processes in human lung tissue
Source: Annual Congress 2010 - Novel cellular mechanisms in lung disease
Year: 2010
Human rhinovirus 16 infection of human macrophages disrupts Arpin expression impairing macrophage internalization: a potential link to exacerbation in chronic obstructive pulmonary disease?
Source: Virtual Congress 2020 – Host-microbe interactions and their influence on lung disease onset and severity
Year: 2020
Mycoplasma pneumoniae induces airway epithelial cell expression of MUC5AC in asthma
Source: Eur Respir J 2008; 31: 43-46
Year: 2008
Egr-1 regulates muc5ac expression through activator protein-1 (AP-1) in chronic airway inflammation diseases
Source: International Congress 2015 – Translational studies in airway cell biology
Year: 2015
Cytokines in chronic obstructive pulmonary disease
Source: Eur Respir J 2001; 18: 50S-59S
Year: 2001
Cigarette smoke inhibits lung fibroblast proliferation by translational mechanisms
Source: Eur Respir J 2012; 39: 705-711
Year: 2012
Epidermal growth factor receptor (EGFR) overexpression in human bronchial epithelial cells amplifies IL-8 release provoked by cigarette smoke and bacterial surrogates
Source: Eur Respir J 2005; 26: Suppl. 49, 441s
Year: 2005
Rhinovirus up-regulates toll-like receptor 3 on bronchial epithelial cells and is dependent on viral replication
Source: Annual Congress 2004 - Mechanisms and treatment of virus induced asthma exacerbations
Year: 2004
Wnt11 inhibits effects of transforming growth factor-beta1 (TGFbeta1) on lung epithelial phenotype
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
Gene polymorphism of epidermal growth factor receptor (EGFR) and airway hyperresponsiveness (AHR) in young allergic subjects without respiratory symptoms
Source: Annual Congress 2011 - Biomarkers of allergic inflammation
Year: 2011
Epidermal growth factor receptor (EGFR) activation is required for TGFbeta1-induced epithelial-mesenchymal transition (EMT) in idiopathic pulmonary fibrosis (IPF)
Source: Annual Congress 2013 –Growth factors at the crossroads of acute and chronic inflammation
Year: 2013
The role of suppressor of cytokine signalling 1 (SOCS1) in virus-induced asthma exacerbations
Source: International Congress 2016 – Viral infections in different respiratory conditions: from the bedside to the lab
Year: 2016
Keratinocyte growth factor improves alterations of lung permeability and bronchial epithelium in allergic rats
Source: Eur Respir J 2007; 30: 31-39
Year: 2007
Induction of mesenchymal cell phenotypes in lung epithelial cells by adenovirus E1A
Source: Eur Respir J 2006; 28: 1106-1117
Year: 2006
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept