Source: Lung Science Conference 2010 - Biology of gene-environment interactions in the lung



Rating:

Share or cite this content

Citations should be made in the following way:
R. Wu, A. Loukoianov, S. Statt, P. Lee, P. Thai, M. Chang (Davis, United States of America). Oral presentation: Epigenetic mechanism involved in smoke-induced persistent elevation of MUC5AC. Lung Science Conference 2010 - Biology of gene-environment interactions in the lung

Member's Comments

No comment yet.

Related content which might interest you: Quercetin attenuates inflammation in cigarette smoke stimulated airway epithelial cells: Possible involvement of autophagy Source: Annual Congress 2013 –New drugs in respiratory medicine Year: 2013 Oral presentation: Altered airway epithelial gene expression determines the lung remodeling response to environmental allergens; role of activin Source: Lung Science Conference 2010 - Biology of gene-environment interactions in the lung Year: 2010 The AMPK-TSC1/2-mTOR signaling pathway regulates cigarette smoking-induced inflammation and apoptosis in airway epithelial cells Source: International Congress 2015 – Molecular pathways of lung injury due to tobacco-related products Year: 2015 Cigarette smoke augments MUC5AC mucin production via TLR3-EGFR pathway in airway epithelial cells Source: Annual Congress 2013 –Stress responses and T-cell behaviour in the lung Year: 2013 Chronic treatment of beta-blocker inhibited mucus hypersecretion and MUC5AC expression exposed to cigarette smoking through beta-arrestin2–ERK1/2 pathway Source: International Congress 2014 – Mechanisms of COPD and inhalation injury Year: 2014 Oral presentation: Genetic or pharmacologic inhibition of ATX stimulates LPS-induced pulmonary inflammation Source: Lung Science Conference 2010 - Biology of gene-environment interactions in the lung Year: 2010 Oral presentation: Role of neutrophil serine proteases in the pathogenesis of cigarette smoke-induced emphysema Source: Lung Science Conference 2010 - Biology of gene-environment interactions in the lung Year: 2010 Oral presentation: Role of CXCL13 in cigarette smoke-induced lymphoid follicle formation and COPD Source: Lung Science Conference 2013 - Early origins and mechanisms of chronic lung disease Year: 2013 Cryptotanshinone attenuates airway Inflammation induced by cigarette smoke in mice Source: International Congress 2016 – New signalling pathways in COPD Year: 2016 Recurrent cigarette smoke exposure impairs SIRT1 shuttling in bronchial epithelial cells Source: International Congress 2016 – Translational studies in lung disease Year: 2016 Cigarette smoke impairs Sirt1 activity and promotes pro-inflammatory responses in bronchial epithelial cells Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects Year: 2015 Role of chronic exposure to cigarette smoke on endoglin/CD105 expression in airway epithelium Source: International Congress 2015 – Cellular and molecular mechanisms of COPD and emphysema Year: 2015 A specific DAMP profile identifies susceptibility to smoke-induced airway inflammation Source: Eur Respir J 2014; 43: 1183-1186 Year: 2014 cPLA2alpha plays a role in neutrophilic inflammatory response in mice induced by tobacco smoke exposure Source: Annual Congress 2013 –Novel in vitro and animal models to study lung diseases Year: 2013 Enhanced inflammatory gene expression in bronchial epithelial cells from asthmatic patients: Attenuation by a Brd4 mimic Source: International Congress 2014 – Novel targets and drugs for asthma and COPD Year: 2014 Ceramide-1-phosphate attenuates cigarette smoke induce lung inflammation Source: Annual Congress 2013 –Cell biology and murine models of asthma and inflammation Year: 2013 Mechanisms of airway epithelial damage: epithelial-mesenchymal interactions in the pathogenesis of asthma Source: Eur Respir J 2003; 22: 24S-29S Year: 2003 Aberrant airway epithelial differentiation resulting from cigarette smoke exposure leads to loss of constitutive antimicrobial peptide and protein (AMP) expression Source: International Congress 2016 – New findings in mucosal immunology Year: 2016 WNT-4 regulates pro-inflammatory responses driven by epithelial-mesenchymal cross-talk Source: International Congress 2016 – Airway pharmacology: drug targets and mode of action Year: 2016