Hypoxia mediated alterations and role of cell cytoskeleton in trafficking

L. Dada (Chicago, United States of America)

Source: Lung Science Conference 2006 - Hypoxia in Lung Biology and Disease
Number: 13

PDF journal article, handout or slides

Rating: 0
You must login to grade this presentation.

Share or cite this content

Citations should be made in the following way:
L. Dada (Chicago, United States of America). Hypoxia mediated alterations and role of cell cytoskeleton in trafficking. Lung Science Conference 2006 - Hypoxia in Lung Biology and Disease

You must login to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.

Member's Comments

No comment yet.
You must Login to comment this presentation.


Related content which might interest you:
Investigating the role of AKAP13 on epithelial cell TGFß activation
Source: International Congress 2019 – New mechanistic insights into pulmonary fibrosis
Year: 2019

Regulation and functions of autophagy
Source: Research Seminar 2014 - Protein quality control in lung disease
Year: 2014

A differential role for ceramide kinase in antigen/FcεRI mediated mast cell activation and function
Source: Eur Respir J 2006; 28: Suppl. 50, 231s
Year: 2006

Chaperone-mediated autophagy modulates epithelial cell apoptosis in COPD pathogenesis.
Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms
Year: 2019

GPR40 promotes tight junction assembly in human airway epithelial cells via AMPK-dependent mechanisms
Source: International Congress 2018 – New kids on the block: novel molecular pathways and therapeutic targets in chronic lung diseases
Year: 2018




Caveolin-1 regulation of thrombin signalling in endothelial cells
Source: Annual Congress 2009 - Acute lung injury: biological mechanisms
Year: 2009


SH2 domain-containing Phosphatase-(SHP)-2 blunts fibrotic responses through regulation of fibroblast mitochondrial metabolism and autophagy
Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease
Year: 2019

PGAM5 acts as a key mediator of lung fibrosis by disrupting mitochondrial homeostasis
Source: International Congress 2019 – Extracellular matrix formation and remodelling in physiology and disease
Year: 2019

Mitochondrial metabolism regulates cellular proteostasis
Source: ERS Lung Science Conference 2020
Year: 2020

Chronic WNT/ß-catenin signaling impairs mitochondrial function in lung epithelial cells
Source: Virtual Congress 2021 – Cellular and molecular involvement in lung injury and repair
Year: 2021



Potential role of filamin and p38 MAPK in the regulation of human alveolar epithelial cell permeability
Source: Eur Respir J 2002; 20: Suppl. 38, 96s
Year: 2002

Human rhinovirus induces the expression of the membrane bound form of its cellular receptor intercellular adhesion molecule 1via a tyrosine kinase dependent signal transduction pathway
Source: Eur Respir J 2001; 18: Suppl. 33, 160s
Year: 2001

miR-146a-5p plays an essential role in the aberrant epithelial–fibroblast cross-talk in COPD
Source: Eur Respir J, 49 (5) 1602538; 10.1183/13993003.02538-2016
Year: 2017



Regulation of cell fate and integrity by mechanical signals
Source: ERS Lung Science Conference 2021
Year: 2021



Cadmium-induced inflammation and ER stress are mediated through C/EBP-DDIT3 signaling in human bronchial epithelial cells
Source: International Congress 2017 – Insight into the pathogenesis of emphysema and pneumonia
Year: 2017

Role of endosomal and cytosolic pattern recognition receptors in dsRNA-induced cytokine expression in human airway smooth muscle cells
Source: Annual Congress 2011 - Airway smooth muscle cells and fibroblasts: cell biology
Year: 2011

Dysregulated mitochondrial integrity associated with lamin B1 reduction is involved in cellular senescence progression in COPD pathogenesis.
Source: International Congress 2018 – New kids on the block: novel molecular pathways and therapeutic targets in chronic lung diseases
Year: 2018



Extracellular vesicles produced by airway epithelial cells in response to oxidative stress contain microRNAs associated with cellular senescence
Source: Virtual Congress 2021 – Cellular and molecular involvement in lung injury and repair
Year: 2021