Theophylline inhibits mobilization of eosinophil induced by interleukin-5 (IL-5) from femoral bone marrow into the blood Source: Annual Congress 2005 - Improving asthma control in the harder-to-reach population Year: 2005
Effect of OVA activated tolerogenic mouse bone marrow dendritic cells on the production of IL-17 in vitro Source: Annual Congress 2013 –Murine models of lung immunology Year: 2013
In vitro effect of shRNA plasmid targeting murine IL-5Rα on IL-5 induced bone marrow eosinopoiesisSource: Eur Respir J 2005; 26: Suppl. 49, 213s Year: 2005
The nasal decongestant oxymetazoline and zinc both inhibit production of pro-inflammatory cytokines in bone marrow-derived mast cells Source: Eur Respir J 2006; 28: Suppl. 50, 577s Year: 2006
Bone marrow derived dendritic cell (BMDC) adoptive transfer alleviate OVA-induced allergic airway inflammation in asthmatic mice Source: Virtual Congress 2020 – New insight into the pathogenesis of chronic lung diseases: asthma, COPD and others Year: 2020
The PBMC of severe asthmatics showed enhanced candida antigen induced IL-5, IL-13 and TARC production and this enhancement was inhibited by dendric cells deprition Source: Annual Congress 2009 - Inflammatory cells and cytokines Year: 2009
IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide- but not allergen-induced airway neutrophilia Source: Eur Respir J 2002; 20: Suppl. 38, 618s Year: 2002
Chemokinome characterization in human lung macrophages following stimulation with Th2-type cytokines IL-4 or IL-13 Source: Annual Congress 2012 - Update on neutrophil and macrophage function in COPD Year: 2012
IL-15 transpresentation in circulating progenitors of NK cells controls their differentiation potential, STAT6 activation and IL-13 production Source: Annual Congress 2005 - Regulation of the allergic and asthmatic response Year: 2005
Overproduction of IL-18 in the lungs induces IL-13 and IFN-g producing CD4+ T cell in the lungs, and results in airway hyperresponsiveness in Balb/c mice Source: Annual Congress 2011 - Asthma: basic science and clinical studies Year: 2011
Pro-inflammatory stimuli affect the production of cytokines and growth factors by human bone marrow-derived mesenchymal stem cells Source: Annual Congress 2008 - Inflammatory trigger mechanisms in asthma and COPD Year: 2008
Airways inflammation and remodeling is inhibited by aerobic exercise via decreases of IL-4 and IL-5 release by inflammatory cells Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007
Streptococcal pneumonia increases the production and release of monocyte from the bone marrow Source: Eur Respir J 2002; 20: Suppl. 38, 228s Year: 2002
LPS-induced TLR4 expression and cytokine release is impaired in monocytes and T-lymphocytes of COPD patients Source: Annual Congress 2008 - Novel targets in the molecular pathology of lung cancer and COPD Year: 2008
IL-10 produced by CD4+ T cells is essential for the development of airway hyperresponsiveness via suppression of endogenous IL-12 Source: Eur Respir J 2005; 26: Suppl. 49, 24s Year: 2005
Aerobic training modulates parenquimal inflammation via decreases of IL-4 and IL-5 release by inflammatory cells Source: Annual Congress 2007 - Towards an understanding of asthma pathogenesis - novel in vitro assays and results from animal models Year: 2007
Regulatory role of antigen-induced IL-10, produced by Tr1 cells, in airway neutrophilia in a murine model for asthma Source: Annual Congress 2012 - Asthma: mechanisms of airway inflammation Year: 2012
Allergen-pulsed bone marrow-derived dendritic cells abolish the influx of allergic inflammatory cells in mouse asthma model Source: Annual Congress 2010 - Animal models of asthma and lung inflammation Year: 2010
Overproduction of mature IL-18 induces IL-13 producing CD4+Tcells in the lungs, and results in airway hyperresponsiveness and pulmonary inflammation in Balb/c mice Source: Annual Congress 2010 - Asthma: basic science and clinical studies Year: 2010
Tim-3 regulates Tregs’ ability to resolve the inflammation and proliferation of acute lung injury by polarizing M2c macrophages Source: International Congress 2017 – Anti-inflammatory and pro-repair mediators in acute lung injury Year: 2017