e-learning
resources
Paris 2018
Sunday, 16.09.2018
Regenerative and anti-fibrotic approaches
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
Cigarette smoke exposure decreases CFLAR expression in bronchial epithelium, augmenting susceptibility for cell death and DAMP release
S. Pouwels (Groningen, Netherlands), A. Faiz (Groningen, Netherlands), I. Heijink (Groningen, Netherlands), C. Vermeulen (Groningen, Netherlands), V. Guryev (Groningen, Netherlands), M. Van Den Berge (Groningen, Netherlands), M. Nawijn (Groningen, Netherlands)
Source:
International Congress 2018 – Regenerative and anti-fibrotic approaches
Session:
Regenerative and anti-fibrotic approaches
Session type:
Poster Discussion
Number:
586
Disease area:
Airway diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
S. Pouwels (Groningen, Netherlands), A. Faiz (Groningen, Netherlands), I. Heijink (Groningen, Netherlands), C. Vermeulen (Groningen, Netherlands), V. Guryev (Groningen, Netherlands), M. Van Den Berge (Groningen, Netherlands), M. Nawijn (Groningen, Netherlands). Cigarette smoke exposure decreases CFLAR expression in bronchial epithelium, augmenting susceptibility for cell death and DAMP release. 586
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Panel discussion: Diagnostic tools for Obstructive Sleep Apnoea in adults and children
Expert interview: Physiological classification of lung function impairment
Occupational exposures to respiratory diseases: A case-based discussion
Related content which might interest you:
Cigarette smoke increases TLR4 expression and modifies LPS mediated responses in airway epithelial cells
Source: Annual Congress 2007 - Epithelial networks in the airways
Year: 2007
Cigarette smoke upregulating the expression of miR 21 increases IL-8 in airway epithelial cells
Source: International Congress 2018 – New kids on the block: novel molecular pathways and therapeutic targets in chronic lung diseases
Year: 2018
Cigarette smoke induces the release of CXCL-8 from human bronchial epithelial cells via TLRs and the induction of inflammasome
Source: Annual Congress 2011 - Epithelial cell biology
Year: 2011
Cigarette smoke induces mitochondrial dysfunction via PKCd-p66Shc signaling pathway in human bronchial epithelial cells
Source: International Congress 2018 – COPD: novel pathological mechanisms in the airway epithelium
Year: 2018
Cigarette smoke impairs Sirt1 activity and promotes pro-inflammatory responses in bronchial epithelial cells
Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects
Year: 2015
Cigarette smoke and TGF-β
1
increase p21
waf1/cip1
expression in alveolar epithelial cells
Source: Eur Respir J 2002; 20: Suppl. 38, 97s
Year: 2002
Cigarette smoke augments MUC5AC mucin production via TLR3-EGFR pathway in airway epithelial cells
Source: Annual Congress 2013 –Stress responses and T-cell behaviour in the lung
Year: 2013
Cigarette smoke exposure disrupts the molecular regulation of mitochondrial metabolism in human bronchial and alveolar epithelial cells
Source: ERS Lung Science Conference 2020
Year: 2020
Cigarette smoke alters the expression of the pro-inflammatory LTB4 receptor and increases the neutrophil adhesion in bronchial epithelial cells
Source: Annual Congress 2011 - Epithelial cell biology
Year: 2011
Recurrent cigarette smoke exposure impairs SIRT1 shuttling in bronchial epithelial cells
Source: International Congress 2016 – Translational studies in lung disease
Year: 2016
Carbocysteine restores the expression and the activation of TLR4 altered by cigarette smoke extracts in bronchial epithelial cells
Source: Annual Congress 2013 –Molecular biology: state-of-the-art 2013
Year: 2013
Cigarette smoke extract induces the release of extracellular vesicles by airway epithelial cells via cellular carbonyl stress
Source: International Congress 2015 – Immunomodulation: basic science and clinical aspects
Year: 2015
Cigarette smoke decreases E-Cadherin expression downregulating FoxO3 in lung epithelial cells
Source: International Congress 2019 – Novel mechanisms elucidated by translational studies of COPD
Year: 2019
Role of chronic exposure to cigarette smoke on endoglin/CD105 expression in airway epithelium
Source: International Congress 2015 – Cellular and molecular mechanisms of COPD and emphysema
Year: 2015
Cigarette smoke modulates rhinovirus-induced airway epithelial cell chemokine production
Source: Eur Respir J 2010; 35: 1256-1263
Year: 2010
The AMPK-TSC1/2-mTOR signaling pathway regulates cigarette smoking-induced inflammation and apoptosis in airway epithelial cells
Source: International Congress 2015 – Molecular pathways of lung injury due to tobacco-related products
Year: 2015
Cigarette smoke suppresses mast cell maturation and cytokine release independent of TLR4 signaling
Source: Annual Congress 2012 - Basic and translational studies in COPD
Year: 2012
Cigarette smoke significantly reduces proliferation and modulates the expression levels of CEBPα and CEBPβ in cultured human lung fibroblasts
Source: Annual Congress 2008 - Basic science in asthma and COPD II
Year: 2008
Cigarette smoking modulates double-stranded RNA-induced interleukin 6 release from bronchial epithelial cells by decreasing TLR-3 expression
Source: Annual Congress 2007 - Immunological function in viral and nonviral pathogens
Year: 2007
Cigarette smoke extract induces barrier changes and senescence in primary bronchial epithelial cells
Source: International Congress 2019 – Effects of environmental exposures in lung pathology and the underlying mechanisms
Year: 2019
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept