e-learning
resources
Copenhagen 2005
Sunday 18.09.2005
From molecules to patients: mechanisms of lung injury, fibrosis and cancer
Login
Search all ERS
e-learning
resources
Disease Areas
Airways Diseases
Interstitial Lung Diseases
Respiratory Critical Care
Respiratory Infections
Paediatric Respiratory Diseases
Pulmonary Vascular Diseases
Sleep and Breathing Disorders
Thoracic Oncology
Events
International Congress
Courses
Webinars
Conferences
Research Seminars
Journal Clubs
Publications
Breathe
Monograph
ERJ
ERJ Open Research
ERR
European Lung White Book
Handbook Series
Guidelines
All ERS guidelines
e-learning
CME Online
Case reports
Short Videos
SpirXpert
Procedure Videos
CME tests
Reference Database of Respiratory Sounds
Radiology Image Challenge
Brief tobacco interventions
EU Projects
VALUE-Dx
ERN-LUNG
ECRAID
UNITE4TB
Disease Areas
Events
Publications
Guidelines
e-learning
EU Projects
Login
Search
The role of alveolar macrophages and type II pneumocytes in enzymatic hydrolysis of dipalmitoyl phosphatidylcholine during bleomycin-induced pulmonary fibrosis
O. Golovach, A. Tahanovich (Minsk, Belarus)
Source:
Annual Congress 2005 - From molecules to patients: mechanisms of lung injury, fibrosis and cancer
Session:
From molecules to patients: mechanisms of lung injury, fibrosis and cancer
Session type:
Thematic Poster Session
Number:
717
Disease area:
Interstitial lung diseases
Rating:
You must
login
to grade this presentation.
Share or cite this content
Citations should be made in the following way:
O. Golovach, A. Tahanovich (Minsk, Belarus). The role of alveolar macrophages and type II pneumocytes in enzymatic hydrolysis of dipalmitoyl phosphatidylcholine during bleomycin-induced pulmonary fibrosis. Eur Respir J 2005; 26: Suppl. 49, 717
You must
login
to share this Presentation/Article on Twitter, Facebook, LinkedIn or by email.
Member's Comments
No comment yet.
You must
Login
to comment this presentation.
Related content which might interest you:
Risk factors associated with the development of interstitial lung abnormalities
Surfactant protein expression at various COPD Gold stages
Dexamethasone inhibits secretory and cytosolic phospholipases A2 in alveolar macrophages in acute lung injury
Related content which might interest you:
Features of enzymatic splitting of phosphatidylcholine dipalmitoyl by alveolar macrophages at a lack of oxygen and the influence of bacterial endotoxins
Source: Eur Respir J 2005; 26: Suppl. 49, 345s
Year: 2005
Defective surfactant biosynthesis and ultrastructural abnormalities of alveolar type 2 cells in pulmonary fibrosis of conditional Nedd4-2mice
Source: International Congress 2019 – Translational aspects of idiopathic pulmonary fibrosis
Year: 2019
The influence of intratracheal surfactant-BL administration or [DCquote]empty“ phosphatidylcholine liposomes on lung surfactant level and alveolar macrophage activity in intact rat lungs
Source: Eur Respir J 2003; 22: Suppl. 45, 73s
Year: 2003
Abnormal intraalveolar accumulation of surfactant proteins in adults with pulmonary alveolar proteinosis
Source: Eur Respir J 2002; 20: Suppl. 38, 90s
Year: 2002
Anionic surfactant phospholipids modulate
Mycoplasma pneumoniae
-induced inflammatory response from alveolar macrophages and U937 cells
Source: Annual Congress 2009 - Mechanisms of infection: what's new?
Year: 2009
Collagen VI-producing macrophages mediate lung fibrosis
Source: International Congress 2019 – Cellular responses and cell therapy in lung injury and repair
Year: 2019
Transplant of alveolar type II cells restore pulmonary surfactant in lung fibrosis
Source: Annual Congress 2010 - Stem cells and growth factors in lung injury
Year: 2010
Increased alveolar soluble annexin V promotes lung inflammation and fibrosis
Source: Eur Respir J 2015; 46: 1417-1429
Year: 2015
Inhibition of pulmonary collagen-I, III and fibrotic cytokins in bleomycin induced pulmonary fibrosis by okam J201
Source: Eur Respir J 2005; 26: Suppl. 49, 81s
Year: 2005
Effect of inhaled surfactant enriched with phosphatidylethanolamine on matrix deposition in a murine model of airway inflammation and remodeling.
Source: International Congress 2018 – Basic pharmacology
Year: 2018
Increased expression of SARS-CoV-2 (Covid-19) adhesion sites on type II pneumocytes, small airway epithelium, and alveolar macrophages in smokers and patients with COPD
Source: Virtual Congress 2021 – Mechanistic pathways in chronic and new lung diseases
Year: 2021
M2 polarized alveolar macrophages in pulmonary fibrosis
Source: Annual Congress 2005 - Pulmonary hypertension and fibrosis: novel mechanisms
Year: 2005
Surfactant proteins in pulmonary alveolar proteinosis in adults
Source: Eur Respir J 2004; 24: 426-435
Year: 2004
Collagen phagocytosis by lung alveolar macrophages in animal models of emphysema
Source: Eur Respir J 2003; 22: 728-734
Year: 2003
Natural lysophospholipids induce antimycobacterial activity in type II alveolar epithelial cells
Source: Annual Congress 2010 - Scientific advances in tuberculosis
Year: 2010
Involvement of the CD40-CD154 interaction in the regulation of surfactant production and development of secondary pulmonary alveolar proteinosis
Source: Annual Congress 2009 - What is new in molecular pathology and functional genomics of neoplastic and non-neoplastic lung disease?
Year: 2009
HSP47siRNA targeting to myofibroblasts attenuates bleomycin-induced pulmonary fibrosis
Source: Annual Congress 2012 - Interstitial lung disease: from bench to bedside
Year: 2012
The effect of tenascin-c deficiency on TGF-β-mediated fibrosis and alveolar inflammation in a murine model of ARDS
Source: Annual Congress 2009 - Pathological remodelling in chronic lung disease: from epithelial-mesenchymal transition to cellular activation
Year: 2009
Lipid peroxidation in cultured rat type II pneumocytes
Source: Eur Respir J 2001; 18: Suppl. 33, 79s
Year: 2001
The role of apoptosis in pulmonary fibrosis
Source: Eur Respir Rev 2008; 17: 138-144
Year: 2008
We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits. By clicking "Accept", you consent to the use of the cookies.
Accept